pubmed-article:19523449 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:19523449 | lifeskim:mentions | umls-concept:C0039194 | lld:lifeskim |
pubmed-article:19523449 | lifeskim:mentions | umls-concept:C1749467 | lld:lifeskim |
pubmed-article:19523449 | lifeskim:mentions | umls-concept:C1332714 | lld:lifeskim |
pubmed-article:19523449 | lifeskim:mentions | umls-concept:C0221117 | lld:lifeskim |
pubmed-article:19523449 | lifeskim:mentions | umls-concept:C0162768 | lld:lifeskim |
pubmed-article:19523449 | lifeskim:mentions | umls-concept:C1622572 | lld:lifeskim |
pubmed-article:19523449 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
pubmed-article:19523449 | lifeskim:mentions | umls-concept:C0332206 | lld:lifeskim |
pubmed-article:19523449 | pubmed:issue | 2 | lld:pubmed |
pubmed-article:19523449 | pubmed:dateCreated | 2009-7-6 | lld:pubmed |
pubmed-article:19523449 | pubmed:abstractText | CD26 binds to caveolin-1 in antigen-presenting cells (APC), and that ligation of CD26 by caveolin-1 induces T cell proliferation in a TCR/CD3-dependent manner. We report herein the effects of CD26-caveolin-1 costimulatory blockade by fusion protein caveolin-1-Ig (Cav-Ig). Soluble Cav-Ig inhibits T cell proliferation and cytokine production in response to recall antigen, or allogeneic APC. Our data hence suggest that blocking of CD26-associated signaling by soluble Cav-Ig may be an effective approach as immunosuppressive therapy. | lld:pubmed |
pubmed-article:19523449 | pubmed:language | eng | lld:pubmed |
pubmed-article:19523449 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19523449 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:19523449 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19523449 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19523449 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19523449 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19523449 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:19523449 | pubmed:month | Aug | lld:pubmed |
pubmed-article:19523449 | pubmed:issn | 1090-2104 | lld:pubmed |
pubmed-article:19523449 | pubmed:author | pubmed-author:MorimotoChika... | lld:pubmed |
pubmed-article:19523449 | pubmed:author | pubmed-author:OhnumaKeiK | lld:pubmed |
pubmed-article:19523449 | pubmed:author | pubmed-author:DangNam HNH | lld:pubmed |
pubmed-article:19523449 | pubmed:author | pubmed-author:UchiyamaMasah... | lld:pubmed |
pubmed-article:19523449 | pubmed:author | pubmed-author:HatanoRyoR | lld:pubmed |
pubmed-article:19523449 | pubmed:author | pubmed-author:EndoYukoY | lld:pubmed |
pubmed-article:19523449 | pubmed:author | pubmed-author:TakasawaWatar... | lld:pubmed |
pubmed-article:19523449 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:19523449 | pubmed:day | 21 | lld:pubmed |
pubmed-article:19523449 | pubmed:volume | 386 | lld:pubmed |
pubmed-article:19523449 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:19523449 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:19523449 | pubmed:pagination | 327-32 | lld:pubmed |
pubmed-article:19523449 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
pubmed-article:19523449 | pubmed:meshHeading | pubmed-meshheading:19523449... | lld:pubmed |
pubmed-article:19523449 | pubmed:meshHeading | pubmed-meshheading:19523449... | lld:pubmed |
pubmed-article:19523449 | pubmed:meshHeading | pubmed-meshheading:19523449... | lld:pubmed |
pubmed-article:19523449 | pubmed:meshHeading | pubmed-meshheading:19523449... | lld:pubmed |
pubmed-article:19523449 | pubmed:meshHeading | pubmed-meshheading:19523449... | lld:pubmed |
pubmed-article:19523449 | pubmed:meshHeading | pubmed-meshheading:19523449... | lld:pubmed |
pubmed-article:19523449 | pubmed:meshHeading | pubmed-meshheading:19523449... | lld:pubmed |
pubmed-article:19523449 | pubmed:meshHeading | pubmed-meshheading:19523449... | lld:pubmed |
pubmed-article:19523449 | pubmed:meshHeading | pubmed-meshheading:19523449... | lld:pubmed |
pubmed-article:19523449 | pubmed:meshHeading | pubmed-meshheading:19523449... | lld:pubmed |
pubmed-article:19523449 | pubmed:year | 2009 | lld:pubmed |
pubmed-article:19523449 | pubmed:articleTitle | Blockade of CD26-mediated T cell costimulation with soluble caveolin-1-Ig fusion protein induces anergy in CD4+T cells. | lld:pubmed |
pubmed-article:19523449 | pubmed:affiliation | Department of Rheumatology and Allergy, Research Hospital, The Institute of Medical Science, The University of Tokyo, Shirokanedai, Tokyo 108-8639, Japan. | lld:pubmed |
pubmed-article:19523449 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:19523449 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |