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pubmed-article:19481913 | lifeskim:mentions | umls-concept:C0439849 | lld:lifeskim |
pubmed-article:19481913 | lifeskim:mentions | umls-concept:C0389071 | lld:lifeskim |
pubmed-article:19481913 | lifeskim:mentions | umls-concept:C0948265 | lld:lifeskim |
pubmed-article:19481913 | lifeskim:mentions | umls-concept:C1384514 | lld:lifeskim |
pubmed-article:19481913 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
pubmed-article:19481913 | lifeskim:mentions | umls-concept:C0085580 | lld:lifeskim |
pubmed-article:19481913 | lifeskim:mentions | umls-concept:C0205419 | lld:lifeskim |
pubmed-article:19481913 | pubmed:issue | 2 | lld:pubmed |
pubmed-article:19481913 | pubmed:dateCreated | 2010-2-15 | lld:pubmed |
pubmed-article:19481913 | pubmed:abstractText | Evidence shows that aldosterone excess is crucial for the development of cardiac and metabolic complications. Among the possible pathogenetic elements of the metabolic syndrome, adiponectin and its polymorphisms seem to confer a genetic risk for metabolic alterations and type 2 diabetes. Aims of the study were to investigate whether metabolic syndrome represents a common feature in patients with primary aldosteronism (PA) compared with essential hypertensives (EH) and to study the impact of two common adiponectin gene variants on the parameters of metabolic syndrome. | lld:pubmed |
pubmed-article:19481913 | pubmed:language | eng | lld:pubmed |
pubmed-article:19481913 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19481913 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:19481913 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:19481913 | pubmed:month | Feb | lld:pubmed |
pubmed-article:19481913 | pubmed:issn | 1590-3729 | lld:pubmed |
pubmed-article:19481913 | pubmed:author | pubmed-author:BoscaroMM | lld:pubmed |
pubmed-article:19481913 | pubmed:author | pubmed-author:GiacchettiGG | lld:pubmed |
pubmed-article:19481913 | pubmed:author | pubmed-author:Di MattiaDD | lld:pubmed |
pubmed-article:19481913 | pubmed:author | pubmed-author:RilliSS | lld:pubmed |
pubmed-article:19481913 | pubmed:author | pubmed-author:AgostinelliLL | lld:pubmed |
pubmed-article:19481913 | pubmed:author | pubmed-author:RonconiVV | lld:pubmed |
pubmed-article:19481913 | pubmed:author | pubmed-author:TurchiFF | lld:pubmed |
pubmed-article:19481913 | pubmed:copyrightInfo | Copyright 2009 Elsevier B.V. All rights reserved. | lld:pubmed |
pubmed-article:19481913 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:19481913 | pubmed:volume | 20 | lld:pubmed |
pubmed-article:19481913 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:19481913 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:19481913 | pubmed:pagination | 93-100 | lld:pubmed |
pubmed-article:19481913 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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pubmed-article:19481913 | pubmed:year | 2010 | lld:pubmed |
pubmed-article:19481913 | pubmed:articleTitle | Metabolic syndrome in primary aldosteronism and essential hypertension: relationship to adiponectin gene variants. | lld:pubmed |
pubmed-article:19481913 | pubmed:affiliation | Division of Endocrinology, Azienda Ospedaliero-Universtaria, Ospedali Riuniti Umberto I-GM Lancisi-G Salesi, Università Politecnica delle Marche, Ancona, Italy. | lld:pubmed |
pubmed-article:19481913 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:19481913 | pubmed:publicationType | Comparative Study | lld:pubmed |
pubmed-article:19481913 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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