pubmed-article:19436669 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:19436669 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
pubmed-article:19436669 | lifeskim:mentions | umls-concept:C0815017 | lld:lifeskim |
pubmed-article:19436669 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
pubmed-article:19436669 | lifeskim:mentions | umls-concept:C0006560 | lld:lifeskim |
pubmed-article:19436669 | lifeskim:mentions | umls-concept:C0333348 | lld:lifeskim |
pubmed-article:19436669 | lifeskim:mentions | umls-concept:C0175630 | lld:lifeskim |
pubmed-article:19436669 | lifeskim:mentions | umls-concept:C0521942 | lld:lifeskim |
pubmed-article:19436669 | lifeskim:mentions | umls-concept:C1637833 | lld:lifeskim |
pubmed-article:19436669 | pubmed:issue | 1 | lld:pubmed |
pubmed-article:19436669 | pubmed:dateCreated | 2009-5-13 | lld:pubmed |
pubmed-article:19436669 | pubmed:abstractText | Anti-inflammatory properties may contribute to the pharmacological effects of angiotensin II receptor blockers (ARBs), a leading therapeutic class in the management of hypertension and related cardiovascular and renal diseases. That possibility, supported by consistent evidence from in-vitro and animal studies showing pro-inflammatory properties of angiotensin II, has been evaluated clinically by measuring the effect of ARBs on C-reactive protein and other circulating indices of inflammation (e-selectin, adhesion molecules, interleukin-6, tissue necrosis factor-alpha, monocyte chemoattractant protein-1) of potential clinical relevance, a body of evidence that this paper aims to review. | lld:pubmed |
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