pubmed-article:19349602 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:19349602 | lifeskim:mentions | umls-concept:C0019080 | lld:lifeskim |
pubmed-article:19349602 | lifeskim:mentions | umls-concept:C0085220 | lld:lifeskim |
pubmed-article:19349602 | lifeskim:mentions | umls-concept:C0021308 | lld:lifeskim |
pubmed-article:19349602 | lifeskim:mentions | umls-concept:C0332281 | lld:lifeskim |
pubmed-article:19349602 | lifeskim:mentions | umls-concept:C0475224 | lld:lifeskim |
pubmed-article:19349602 | lifeskim:mentions | umls-concept:C2828008 | lld:lifeskim |
pubmed-article:19349602 | pubmed:issue | 14 | lld:pubmed |
pubmed-article:19349602 | pubmed:dateCreated | 2009-4-7 | lld:pubmed |
pubmed-article:19349602 | pubmed:abstractText | Neuropathologic studies suggest an association between cerebral amyloid angiopathy (CAA) and small ischemic infarctions as well as hemorrhages. We examined the prevalence and associated risk factors for infarcts detected by diffusion-weighted imaging (DWI). | lld:pubmed |
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pubmed-article:19349602 | pubmed:language | eng | lld:pubmed |
pubmed-article:19349602 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19349602 | pubmed:citationSubset | AIM | lld:pubmed |
pubmed-article:19349602 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:19349602 | pubmed:month | Apr | lld:pubmed |
pubmed-article:19349602 | pubmed:issn | 1526-632X | lld:pubmed |
pubmed-article:19349602 | pubmed:author | pubmed-author:SmithE EEE | lld:pubmed |
pubmed-article:19349602 | pubmed:author | pubmed-author:GreenbergS... | lld:pubmed |
pubmed-article:19349602 | pubmed:author | pubmed-author:GilsonAA | lld:pubmed |
pubmed-article:19349602 | pubmed:author | pubmed-author:ViswanathanAA | lld:pubmed |
pubmed-article:19349602 | pubmed:author | pubmed-author:RossN TNT | lld:pubmed |
pubmed-article:19349602 | pubmed:author | pubmed-author:RosanoGG | lld:pubmed |
pubmed-article:19349602 | pubmed:author | pubmed-author:KimberlyW TWT | lld:pubmed |
pubmed-article:19349602 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:19349602 | pubmed:day | 7 | lld:pubmed |
pubmed-article:19349602 | pubmed:volume | 72 | lld:pubmed |
pubmed-article:19349602 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:19349602 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:19349602 | pubmed:pagination | 1230-5 | lld:pubmed |
pubmed-article:19349602 | pubmed:dateRevised | 2010-9-27 | lld:pubmed |
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pubmed-article:19349602 | pubmed:year | 2009 | lld:pubmed |
pubmed-article:19349602 | pubmed:articleTitle | Silent ischemic infarcts are associated with hemorrhage burden in cerebral amyloid angiopathy. | lld:pubmed |
pubmed-article:19349602 | pubmed:affiliation | Hemorrhagic Stroke Research Program, Department of Neurology, Massachusetts General Hospital and Harvard Medical School, Boston, MA, USA. | lld:pubmed |
pubmed-article:19349602 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:19349602 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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