| pubmed-article:19348925 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19348925 | lifeskim:mentions | umls-concept:C0221912 | lld:lifeskim |
| pubmed-article:19348925 | lifeskim:mentions | umls-concept:C0754728 | lld:lifeskim |
| pubmed-article:19348925 | lifeskim:mentions | umls-concept:C1704632 | lld:lifeskim |
| pubmed-article:19348925 | lifeskim:mentions | umls-concept:C0871261 | lld:lifeskim |
| pubmed-article:19348925 | lifeskim:mentions | umls-concept:C1325847 | lld:lifeskim |
| pubmed-article:19348925 | lifeskim:mentions | umls-concept:C2911692 | lld:lifeskim |
| pubmed-article:19348925 | lifeskim:mentions | umls-concept:C1706817 | lld:lifeskim |
| pubmed-article:19348925 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:19348925 | pubmed:dateCreated | 2009-4-7 | lld:pubmed |
| pubmed-article:19348925 | pubmed:abstractText | Atopic dermatitis and allergic contact dermatitis are skin disorders triggered by epicutaneous sensitization with protein antigens and contact sensitization with haptens, respectively. Skin is colonized with bacteria, which are a source of Toll-like receptor (TLR) 2 ligands. | lld:pubmed |
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| pubmed-article:19348925 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:19348925 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:19348925 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19348925 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:19348925 | pubmed:issn | 1097-6825 | lld:pubmed |
| pubmed-article:19348925 | pubmed:author | pubmed-author:ZurakowskiDav... | lld:pubmed |
| pubmed-article:19348925 | pubmed:author | pubmed-author:KumarLalitL | lld:pubmed |
| pubmed-article:19348925 | pubmed:author | pubmed-author:OettgenHansH | lld:pubmed |
| pubmed-article:19348925 | pubmed:author | pubmed-author:GorelikLeonid... | lld:pubmed |
| pubmed-article:19348925 | pubmed:author | pubmed-author:GehaRaifR | lld:pubmed |
| pubmed-article:19348925 | pubmed:author | pubmed-author:JinHaoliH | lld:pubmed |
| pubmed-article:19348925 | pubmed:author | pubmed-author:MathiasClinto... | lld:pubmed |
| pubmed-article:19348925 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:19348925 | pubmed:volume | 123 | lld:pubmed |
| pubmed-article:19348925 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19348925 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19348925 | pubmed:pagination | 875-82.e1 | lld:pubmed |
| pubmed-article:19348925 | pubmed:dateRevised | 2011-9-26 | lld:pubmed |
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| pubmed-article:19348925 | pubmed:meshHeading | pubmed-meshheading:19348925... | lld:pubmed |
| pubmed-article:19348925 | pubmed:meshHeading | pubmed-meshheading:19348925... | lld:pubmed |
| pubmed-article:19348925 | pubmed:meshHeading | pubmed-meshheading:19348925... | lld:pubmed |
| pubmed-article:19348925 | pubmed:meshHeading | pubmed-meshheading:19348925... | lld:pubmed |
| pubmed-article:19348925 | pubmed:meshHeading | pubmed-meshheading:19348925... | lld:pubmed |
| pubmed-article:19348925 | pubmed:meshHeading | pubmed-meshheading:19348925... | lld:pubmed |
| pubmed-article:19348925 | pubmed:meshHeading | pubmed-meshheading:19348925... | lld:pubmed |
| pubmed-article:19348925 | pubmed:meshHeading | pubmed-meshheading:19348925... | lld:pubmed |
| pubmed-article:19348925 | pubmed:meshHeading | pubmed-meshheading:19348925... | lld:pubmed |
| pubmed-article:19348925 | pubmed:year | 2009 | lld:pubmed |
| pubmed-article:19348925 | pubmed:articleTitle | Toll-like receptor 2 is important for the T(H)1 response to cutaneous sensitization. | lld:pubmed |
| pubmed-article:19348925 | pubmed:affiliation | Division of Immunology, Children's Hospital, and the Department of Pediatrics, Harvard Medical School, Boston, MA 02115, USA. | lld:pubmed |
| pubmed-article:19348925 | pubmed:publicationType | Journal Article | lld:pubmed |
