pubmed-article:19327134 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:19327134 | lifeskim:mentions | umls-concept:C0035647 | lld:lifeskim |
pubmed-article:19327134 | lifeskim:mentions | umls-concept:C0332307 | lld:lifeskim |
pubmed-article:19327134 | lifeskim:mentions | umls-concept:C0011860 | lld:lifeskim |
pubmed-article:19327134 | lifeskim:mentions | umls-concept:C0010068 | lld:lifeskim |
pubmed-article:19327134 | lifeskim:mentions | umls-concept:C1848822 | lld:lifeskim |
pubmed-article:19327134 | lifeskim:mentions | umls-concept:C0597357 | lld:lifeskim |
pubmed-article:19327134 | lifeskim:mentions | umls-concept:C0003017 | lld:lifeskim |
pubmed-article:19327134 | lifeskim:mentions | umls-concept:C0032529 | lld:lifeskim |
pubmed-article:19327134 | pubmed:dateCreated | 2009-4-15 | lld:pubmed |
pubmed-article:19327134 | pubmed:abstractText | Increased activation of the renin-angiotensin system (RAS) may be important in promoting coronary heart disease (CHD) and renal dysfunction, but limited data are available on associations between angiotensin type 1 receptor (AGT1R) and angiotensinogen (AGT) genotypes in type 2 diabetes. | lld:pubmed |
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pubmed-article:19327134 | pubmed:language | eng | lld:pubmed |
pubmed-article:19327134 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19327134 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:19327134 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:19327134 | pubmed:issn | 1471-2369 | lld:pubmed |
pubmed-article:19327134 | pubmed:author | pubmed-author:CoxF WFW | lld:pubmed |
pubmed-article:19327134 | pubmed:author | pubmed-author:HuFrank BFB | lld:pubmed |
pubmed-article:19327134 | pubmed:author | pubmed-author:CurhanGary... | lld:pubmed |
pubmed-article:19327134 | pubmed:author | pubmed-author:LinJulieJ | lld:pubmed |
pubmed-article:19327134 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:19327134 | pubmed:volume | 10 | lld:pubmed |
pubmed-article:19327134 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:19327134 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:19327134 | pubmed:pagination | 9 | lld:pubmed |
pubmed-article:19327134 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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pubmed-article:19327134 | pubmed:year | 2009 | lld:pubmed |
pubmed-article:19327134 | pubmed:articleTitle | Genetic polymorphisms of angiotensin-2 type 1 receptor and angiotensinogen and risk of renal dysfunction and coronary heart disease in type 2 diabetes mellitus. | lld:pubmed |
pubmed-article:19327134 | pubmed:affiliation | Channing Laboratory, Department of Medicine, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA. jlin11@partners.org | lld:pubmed |
pubmed-article:19327134 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:19327134 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |