| pubmed-article:19252134 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19252134 | lifeskim:mentions | umls-concept:C0011854 | lld:lifeskim |
| pubmed-article:19252134 | lifeskim:mentions | umls-concept:C0919453 | lld:lifeskim |
| pubmed-article:19252134 | lifeskim:mentions | umls-concept:C0011881 | lld:lifeskim |
| pubmed-article:19252134 | lifeskim:mentions | umls-concept:C0004083 | lld:lifeskim |
| pubmed-article:19252134 | lifeskim:mentions | umls-concept:C0034606 | lld:lifeskim |
| pubmed-article:19252134 | lifeskim:mentions | umls-concept:C0441633 | lld:lifeskim |
| pubmed-article:19252134 | pubmed:issue | 6 | lld:pubmed |
| pubmed-article:19252134 | pubmed:dateCreated | 2009-5-22 | lld:pubmed |
| pubmed-article:19252134 | pubmed:abstractText | Despite extensive evidence for genetic susceptibility to diabetic nephropathy, the identification of susceptibility genes and their variants has had limited success. To search for genes that contribute to diabetic nephropathy, a genome-wide association scan was implemented on the Genetics of Kidneys in Diabetes collection. | lld:pubmed |
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| pubmed-article:19252134 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:19252134 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:19252134 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19252134 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:19252134 | pubmed:issn | 1939-327X | lld:pubmed |
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| pubmed-article:19252134 | pubmed:author | pubmed-author:KleinJon BJB | lld:pubmed |
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| pubmed-article:19252134 | pubmed:author | pubmed-author:PezzolesiMarc... | lld:pubmed |
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| pubmed-article:19252134 | pubmed:author | pubmed-author:WolkowPawelP | lld:pubmed |
| pubmed-article:19252134 | pubmed:author | pubmed-author:WaggottDarylD | lld:pubmed |
| pubmed-article:19252134 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:19252134 | pubmed:volume | 58 | lld:pubmed |
| pubmed-article:19252134 | pubmed:owner | NLM | lld:pubmed |
