| pubmed-article:19237300 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19237300 | lifeskim:mentions | umls-concept:C0038435 | lld:lifeskim |
| pubmed-article:19237300 | lifeskim:mentions | umls-concept:C0226890 | lld:lifeskim |
| pubmed-article:19237300 | lifeskim:mentions | umls-concept:C0021390 | lld:lifeskim |
| pubmed-article:19237300 | lifeskim:mentions | umls-concept:C0014239 | lld:lifeskim |
| pubmed-article:19237300 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:19237300 | pubmed:dateCreated | 2009-6-1 | lld:pubmed |
| pubmed-article:19237300 | pubmed:abstractText | The unfolded protein response as a consequence of endoplasmic reticulum (ER) stress has recently been implicated as a novel mechanism that may lead to inflammatory bowel disease (IBD). Impairment of proper ER stress resolution in highly secretory Paneth and, to a lesser extent, goblet cells within the epithelium can primarily lead to intestinal inflammation. An inability to manage ER stress may not only be a primary originator of intestinal inflammation as exemplified by genetic polymorphisms in XBP1 that are associated with IBD but also a perpetuator of inflammation when ER stress is induced secondarily to inflammatory mediators or microbial factors. Furthermore, ER stress pathways may interact with other processes that lead to IBD, notably autophagy. | lld:pubmed |
| pubmed-article:19237300 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19237300 | pubmed:language | eng | lld:pubmed |
| pubmed-article:19237300 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19237300 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:19237300 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19237300 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19237300 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:19237300 | pubmed:issn | 1096-3618 | lld:pubmed |
| pubmed-article:19237300 | pubmed:author | pubmed-author:BlumbergRicha... | lld:pubmed |
| pubmed-article:19237300 | pubmed:author | pubmed-author:KaserArthurA | lld:pubmed |
| pubmed-article:19237300 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:19237300 | pubmed:volume | 21 | lld:pubmed |
| pubmed-article:19237300 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19237300 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19237300 | pubmed:pagination | 156-63 | lld:pubmed |
| pubmed-article:19237300 | pubmed:dateRevised | 2011-3-4 | lld:pubmed |
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| pubmed-article:19237300 | pubmed:meshHeading | pubmed-meshheading:19237300... | lld:pubmed |
| pubmed-article:19237300 | pubmed:year | 2009 | lld:pubmed |
| pubmed-article:19237300 | pubmed:articleTitle | Endoplasmic reticulum stress in the intestinal epithelium and inflammatory bowel disease. | lld:pubmed |
| pubmed-article:19237300 | pubmed:affiliation | Department of Medicine II (Gastroenterology and Hepatology), Innsbruck Medical University, Innsbruck, Austria. | lld:pubmed |
| pubmed-article:19237300 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:19237300 | pubmed:publicationType | Review | lld:pubmed |
| entrez-gene:7494 | entrezgene:pubmed | pubmed-article:19237300 | lld:entrezgene |
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