pubmed-article:19198651 | rdf:type | pubmed:Citation | lld:pubmed |
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pubmed-article:19198651 | lifeskim:mentions | umls-concept:C2698600 | lld:lifeskim |
pubmed-article:19198651 | lifeskim:mentions | umls-concept:C2911692 | lld:lifeskim |
pubmed-article:19198651 | lifeskim:mentions | umls-concept:C1706817 | lld:lifeskim |
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pubmed-article:19198651 | lifeskim:mentions | umls-concept:C0205349 | lld:lifeskim |
pubmed-article:19198651 | lifeskim:mentions | umls-concept:C1708936 | lld:lifeskim |
pubmed-article:19198651 | lifeskim:mentions | umls-concept:C0301625 | lld:lifeskim |
pubmed-article:19198651 | pubmed:issue | 2 | lld:pubmed |
pubmed-article:19198651 | pubmed:dateCreated | 2009-2-9 | lld:pubmed |
pubmed-article:19198651 | pubmed:abstractText | The relationship of elevated T cell activation to altered T cell differentiation profiles, each defining features of HIV-1 infection, has not been extensively explored. We hypothesized that anti-retroviral suppression of T cell activation levels would lead to alterations in the T cell differentiation of total and HIV-1 specific CD8+ T cell responses among recently HIV-1 infected adults. | lld:pubmed |
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pubmed-article:19198651 | pubmed:language | eng | lld:pubmed |
pubmed-article:19198651 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19198651 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:19198651 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:19198651 | pubmed:issn | 1932-6203 | lld:pubmed |
pubmed-article:19198651 | pubmed:author | pubmed-author:BarbourJason... | lld:pubmed |
pubmed-article:19198651 | pubmed:author | pubmed-author:HechtFrederic... | lld:pubmed |
pubmed-article:19198651 | pubmed:author | pubmed-author:LevyJay AJA | lld:pubmed |
pubmed-article:19198651 | pubmed:author | pubmed-author:SinclairEliza... | lld:pubmed |
pubmed-article:19198651 | pubmed:author | pubmed-author:NdhlovuLishom... | lld:pubmed |
pubmed-article:19198651 | pubmed:author | pubmed-author:BredtBarry... | lld:pubmed |
pubmed-article:19198651 | pubmed:author | pubmed-author:EplingLorrieL | lld:pubmed |
pubmed-article:19198651 | pubmed:author | pubmed-author:HoTerenceT | lld:pubmed |
pubmed-article:19198651 | pubmed:author | pubmed-author:Xuan TanQiQ | lld:pubmed |
pubmed-article:19198651 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:19198651 | pubmed:volume | 4 | lld:pubmed |
pubmed-article:19198651 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:19198651 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:19198651 | pubmed:pagination | e4408 | lld:pubmed |
pubmed-article:19198651 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
pubmed-article:19198651 | pubmed:meshHeading | pubmed-meshheading:19198651... | lld:pubmed |
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pubmed-article:19198651 | pubmed:meshHeading | pubmed-meshheading:19198651... | lld:pubmed |
pubmed-article:19198651 | pubmed:year | 2009 | lld:pubmed |
pubmed-article:19198651 | pubmed:articleTitle | High CD8+ T cell activation marks a less differentiated HIV-1 specific CD8+ T cell response that is not altered by suppression of viral replication. | lld:pubmed |
pubmed-article:19198651 | pubmed:affiliation | Department of Medicine, HIV/AIDS Division, San Francisco General Hospital, University of California San Francisco, San Francisco, California, United States of America. jbarbour@php.ucsf.edu | lld:pubmed |
pubmed-article:19198651 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:19198651 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
pubmed-article:19198651 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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