| pubmed-article:19197984 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19197984 | lifeskim:mentions | umls-concept:C0035647 | lld:lifeskim |
| pubmed-article:19197984 | lifeskim:mentions | umls-concept:C0442027 | lld:lifeskim |
| pubmed-article:19197984 | lifeskim:mentions | umls-concept:C0387583 | lld:lifeskim |
| pubmed-article:19197984 | lifeskim:mentions | umls-concept:C0850639 | lld:lifeskim |
| pubmed-article:19197984 | lifeskim:mentions | umls-concept:C0392756 | lld:lifeskim |
| pubmed-article:19197984 | lifeskim:mentions | umls-concept:C0678951 | lld:lifeskim |
| pubmed-article:19197984 | pubmed:issue | 7 | lld:pubmed |
| pubmed-article:19197984 | pubmed:dateCreated | 2009-3-25 | lld:pubmed |
| pubmed-article:19197984 | pubmed:abstractText | Oral premalignant lesions (OPLs) have the potential to transform into malignant oral cancers. Overexpression of the cyclooxygenase-2 gene (COX-2) is observed frequently in OPLs and oral cancers, suggesting that this gene may play an important role in the progression of oral cancer. Single-nucleotide polymorphisms of COX-2 have been associated with the risk of multiple cancers; however, to date, their effects on OPL susceptibility have not been evaluated sufficiently. | lld:pubmed |
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| pubmed-article:19197984 | pubmed:language | eng | lld:pubmed |
| pubmed-article:19197984 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19197984 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:19197984 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19197984 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:19197984 | pubmed:issn | 0008-543X | lld:pubmed |
| pubmed-article:19197984 | pubmed:author | pubmed-author:XiaPuP | lld:pubmed |
| pubmed-article:19197984 | pubmed:author | pubmed-author:LeeJ JackJJ | lld:pubmed |
| pubmed-article:19197984 | pubmed:author | pubmed-author:LippmanScott... | lld:pubmed |
| pubmed-article:19197984 | pubmed:author | pubmed-author:WuXifengX | lld:pubmed |
| pubmed-article:19197984 | pubmed:author | pubmed-author:YangHushanH | lld:pubmed |
| pubmed-article:19197984 | pubmed:copyrightInfo | (c) 2009 American Cancer Society | lld:pubmed |
| pubmed-article:19197984 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:19197984 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:19197984 | pubmed:volume | 115 | lld:pubmed |
| pubmed-article:19197984 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19197984 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19197984 | pubmed:pagination | 1498-506 | lld:pubmed |
| pubmed-article:19197984 | pubmed:dateRevised | 2010-12-17 | lld:pubmed |
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| pubmed-article:19197984 | pubmed:year | 2009 | lld:pubmed |
| pubmed-article:19197984 | pubmed:articleTitle | Cyclooxygenase-2 gene polymorphisms reduce the risk of oral premalignant lesions. | lld:pubmed |
| pubmed-article:19197984 | pubmed:affiliation | Department of Epidemiology, The University of Texas M. D. Anderson Cancer Center, Houston, Texas 77030, USA. | lld:pubmed |
| pubmed-article:19197984 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:19197984 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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