pubmed-article:19174497 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:19174497 | lifeskim:mentions | umls-concept:C0030705 | lld:lifeskim |
pubmed-article:19174497 | lifeskim:mentions | umls-concept:C0332307 | lld:lifeskim |
pubmed-article:19174497 | lifeskim:mentions | umls-concept:C0011860 | lld:lifeskim |
pubmed-article:19174497 | lifeskim:mentions | umls-concept:C0020615 | lld:lifeskim |
pubmed-article:19174497 | lifeskim:mentions | umls-concept:C2349975 | lld:lifeskim |
pubmed-article:19174497 | lifeskim:mentions | umls-concept:C1570906 | lld:lifeskim |
pubmed-article:19174497 | pubmed:issue | 4 | lld:pubmed |
pubmed-article:19174497 | pubmed:dateCreated | 2009-4-7 | lld:pubmed |
pubmed-article:19174497 | pubmed:databankReference | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19174497 | pubmed:abstractText | Dipeptidyl peptidase-4 inhibitors act by increasing plasma levels of glucagon-like peptide-1 and suppressing excessive glucagon secretion in patients with type 2 diabetes. However, their effects on the glucagon response to hypoglycemia are not established. | lld:pubmed |
pubmed-article:19174497 | pubmed:language | eng | lld:pubmed |
pubmed-article:19174497 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19174497 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:19174497 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:19174497 | pubmed:month | Apr | lld:pubmed |
pubmed-article:19174497 | pubmed:issn | 1945-7197 | lld:pubmed |
pubmed-article:19174497 | pubmed:author | pubmed-author:AhrénBoB | lld:pubmed |
pubmed-article:19174497 | pubmed:author | pubmed-author:NilssonPeter... | lld:pubmed |
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pubmed-article:19174497 | pubmed:author | pubmed-author:FoleyJames... | lld:pubmed |
pubmed-article:19174497 | pubmed:author | pubmed-author:SchweizerAnja... | lld:pubmed |
pubmed-article:19174497 | pubmed:author | pubmed-author:DejagerSylvie... | lld:pubmed |
pubmed-article:19174497 | pubmed:author | pubmed-author:PerssonMargar... | lld:pubmed |
pubmed-article:19174497 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:19174497 | pubmed:volume | 94 | lld:pubmed |
pubmed-article:19174497 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:19174497 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:19174497 | pubmed:pagination | 1236-43 | lld:pubmed |
pubmed-article:19174497 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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pubmed-article:19174497 | pubmed:year | 2009 | lld:pubmed |
pubmed-article:19174497 | pubmed:articleTitle | Vildagliptin enhances islet responsiveness to both hyper- and hypoglycemia in patients with type 2 diabetes. | lld:pubmed |
pubmed-article:19174497 | pubmed:affiliation | Department of Clinical Sciences, Lund University, Lund, Sweden. bo.ahren@med.lu.se | lld:pubmed |
pubmed-article:19174497 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:19174497 | pubmed:publicationType | Randomized Controlled Trial | lld:pubmed |
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