pubmed-article:19095731 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:19095731 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
pubmed-article:19095731 | lifeskim:mentions | umls-concept:C0006560 | lld:lifeskim |
pubmed-article:19095731 | pubmed:issue | 2 | lld:pubmed |
pubmed-article:19095731 | pubmed:dateCreated | 2009-1-30 | lld:pubmed |
pubmed-article:19095731 | pubmed:abstractText | Inflammation is pivotal in all phases of atherosclerosis. Among the numerous inflammatory biomarkers, the largest amount of published data supports a role for C-reactive protein (CRP) as a robust and independent risk marker in the prediction of primary and secondary adverse cardiovascular events. In addition to being a risk marker, there is much evidence indicating that CRP may indeed participate in atherogenesis. | lld:pubmed |
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pubmed-article:19095731 | pubmed:language | eng | lld:pubmed |
pubmed-article:19095731 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19095731 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:19095731 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19095731 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19095731 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:19095731 | pubmed:month | Feb | lld:pubmed |
pubmed-article:19095731 | pubmed:issn | 1530-8561 | lld:pubmed |
pubmed-article:19095731 | pubmed:author | pubmed-author:JialalIshwarl... | lld:pubmed |
pubmed-article:19095731 | pubmed:author | pubmed-author:DevarajSridev... | lld:pubmed |