| pubmed-article:1904009 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:1904009 | lifeskim:mentions | umls-concept:C0025936 | lld:lifeskim |
| pubmed-article:1904009 | lifeskim:mentions | umls-concept:C0598766 | lld:lifeskim |
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| pubmed-article:1904009 | lifeskim:mentions | umls-concept:C2827395 | lld:lifeskim |
| pubmed-article:1904009 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:1904009 | pubmed:dateCreated | 1991-7-5 | lld:pubmed |
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| pubmed-article:1904009 | pubmed:abstractText | To search for genes that can collaborate with myc in lymphomagenesis, we exploited retroviral insertional mutagenesis in E mu-myc transgenic mice. Moloney murine leukemia virus accelerated development of B lymphoid tumors. Three quarters contained a provirus within the known pim-1 or pim-2 loci, new loci bmi-1 and emi-1, or combinations of these. bmi-1 insertions predominated, occurring in half the tumors, and resulted in elevated bmi-1 mRNA levels. Significantly, the bmi-1 gene, which is expressed in diverse normal cells, encodes a Cys/His metal-binding motif (C3HC4) that resembles those in several DNA-binding proteins and defines a new category of zinc finger gene. Thus, myc-induced lymphomagenesis can entail the concerted action of several genes, including the presumptive nuclear regulator bmi-1. | lld:pubmed |
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| pubmed-article:1904009 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:1904009 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:1904009 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1904009 | pubmed:month | May | lld:pubmed |
| pubmed-article:1904009 | pubmed:issn | 0092-8674 | lld:pubmed |
| pubmed-article:1904009 | pubmed:author | pubmed-author:AdamsJ MJM | lld:pubmed |
| pubmed-article:1904009 | pubmed:author | pubmed-author:AlexanderW... | lld:pubmed |
| pubmed-article:1904009 | pubmed:author | pubmed-author:KlinkenS PSP | lld:pubmed |
| pubmed-article:1904009 | pubmed:author | pubmed-author:BarryWW | lld:pubmed |
| pubmed-article:1904009 | pubmed:author | pubmed-author:HauptYY | lld:pubmed |
| pubmed-article:1904009 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1904009 | pubmed:day | 31 | lld:pubmed |
| pubmed-article:1904009 | pubmed:volume | 65 | lld:pubmed |
| pubmed-article:1904009 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1904009 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1904009 | pubmed:pagination | 753-63 | lld:pubmed |
| pubmed-article:1904009 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:1904009 | pubmed:year | 1991 | lld:pubmed |
| pubmed-article:1904009 | pubmed:articleTitle | Novel zinc finger gene implicated as myc collaborator by retrovirally accelerated lymphomagenesis in E mu-myc transgenic mice. | lld:pubmed |
| pubmed-article:1904009 | pubmed:affiliation | Walter and Eliza Hall Institute of Medical Research, P.O. Royal Melbourne Hospital, Victoria, Australia. | lld:pubmed |
| pubmed-article:1904009 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:1904009 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:1904009 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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