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pubmed-article:18824744pubmed:abstractTextThe inability of skeletal myoblasts to transdifferentiate into cardiomyocytes suggests that their beneficial effects on cardiac function after a myocardial infarction are mediated by paracrine effects. We evaluated the roles of these factors in the preservation of matrix architecture (in the infarct and remote regions) by varying the timing (postmyocardial infarction) and delivery site of the implanted cells.lld:pubmed
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pubmed-article:18824744pubmed:articleTitleSkeletal myoblasts preserve remote matrix architecture and global function when implanted early or late after coronary ligation into infarcted or remote myocardium.lld:pubmed
pubmed-article:18824744pubmed:affiliationMaRS Centre, Toronto Medical Discovery Tower, Room #3-702, 101 College Street, Toronto, Ontario, Canada.lld:pubmed
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