| pubmed-article:18802014 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18802014 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:18802014 | lifeskim:mentions | umls-concept:C0040053 | lld:lifeskim |
| pubmed-article:18802014 | lifeskim:mentions | umls-concept:C0042971 | lld:lifeskim |
| pubmed-article:18802014 | lifeskim:mentions | umls-concept:C0134835 | lld:lifeskim |
| pubmed-article:18802014 | lifeskim:mentions | umls-concept:C0376618 | lld:lifeskim |
| pubmed-article:18802014 | lifeskim:mentions | umls-concept:C0443199 | lld:lifeskim |
| pubmed-article:18802014 | pubmed:issue | 12 | lld:pubmed |
| pubmed-article:18802014 | pubmed:dateCreated | 2008-11-21 | lld:pubmed |
| pubmed-article:18802014 | pubmed:abstractText | Endotoxin (lipopolysaccharide [LPS]) enhances microvascular thrombosis in mouse cremaster venules. Because von Willebrand factor (vWF) and P-selectin are suggested to mediate LPS-induced platelet-microvessel interactions, we determined whether vWF and P-selectin contribute to microvascular thrombosis in endotoxemia. | lld:pubmed |
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| pubmed-article:18802014 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:18802014 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:18802014 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18802014 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:18802014 | pubmed:issn | 1524-4636 | lld:pubmed |
| pubmed-article:18802014 | pubmed:author | pubmed-author:McMullenColle... | lld:pubmed |
| pubmed-article:18802014 | pubmed:author | pubmed-author:BurnsAlan RAR | lld:pubmed |
| pubmed-article:18802014 | pubmed:author | pubmed-author:DongJing-FeiJ... | lld:pubmed |
| pubmed-article:18802014 | pubmed:author | pubmed-author:RumbautRoland... | lld:pubmed |
| pubmed-article:18802014 | pubmed:author | pubmed-author:SoubraSaid... | lld:pubmed |
| pubmed-article:18802014 | pubmed:author | pubmed-author:BelleraRicard... | lld:pubmed |
| pubmed-article:18802014 | pubmed:author | pubmed-author:PatelKavita... | lld:pubmed |
| pubmed-article:18802014 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18802014 | pubmed:volume | 28 | lld:pubmed |
| pubmed-article:18802014 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18802014 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18802014 | pubmed:pagination | 2225-30 | lld:pubmed |
| pubmed-article:18802014 | pubmed:dateRevised | 2011-9-26 | lld:pubmed |
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| pubmed-article:18802014 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18802014 | pubmed:articleTitle | Differential role of von Willebrand factor and P-selectin on microvascular thrombosis in endotoxemia. | lld:pubmed |
| pubmed-article:18802014 | pubmed:affiliation | Medical Care Line, Michael E. DeBakey VA Medical Center, Houston, TX, USA. | lld:pubmed |
| pubmed-article:18802014 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18802014 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
| pubmed-article:18802014 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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