| pubmed-article:18725054 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18725054 | lifeskim:mentions | umls-concept:C0011860 | lld:lifeskim |
| pubmed-article:18725054 | lifeskim:mentions | umls-concept:C0009566 | lld:lifeskim |
| pubmed-article:18725054 | lifeskim:mentions | umls-concept:C0814299 | lld:lifeskim |
| pubmed-article:18725054 | lifeskim:mentions | umls-concept:C1527148 | lld:lifeskim |
| pubmed-article:18725054 | lifeskim:mentions | umls-concept:C2825097 | lld:lifeskim |
| pubmed-article:18725054 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
| pubmed-article:18725054 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:18725054 | pubmed:dateCreated | 2008-8-26 | lld:pubmed |
| pubmed-article:18725054 | pubmed:abstractText | Diabetes is characterized by chronic hyperglycemia that produces dysregulation of cellular metabolism and result in excess free radical production and oxidative stress. Glutathione S-transferases (GSTs) are a family of multifunctional enzymes that work as antioxidants. Therefore, diminished expression of GSTs may result in a reduced body defense against oxidative stress, followed by development of diabetic complications. | lld:pubmed |
| pubmed-article:18725054 | pubmed:language | eng | lld:pubmed |
| pubmed-article:18725054 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18725054 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:18725054 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18725054 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18725054 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:18725054 | pubmed:issn | 1557-8615 | lld:pubmed |
| pubmed-article:18725054 | pubmed:author | pubmed-author:HossainiAyshe... | lld:pubmed |
| pubmed-article:18725054 | pubmed:author | pubmed-author:ZamrroniIsraa... | lld:pubmed |
| pubmed-article:18725054 | pubmed:author | pubmed-author:KashemReem... | lld:pubmed |
| pubmed-article:18725054 | pubmed:author | pubmed-author:KhanZoiya F... | lld:pubmed |
| pubmed-article:18725054 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18725054 | pubmed:volume | 23 | lld:pubmed |
| pubmed-article:18725054 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18725054 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18725054 | pubmed:pagination | 444-8 | lld:pubmed |
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| pubmed-article:18725054 | pubmed:meshHeading | pubmed-meshheading:18725054... | lld:pubmed |
| pubmed-article:18725054 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18725054 | pubmed:articleTitle | Polymorphism of glutathione S-transferases as genetic risk factors for the development of complications in type 2 diabetes mellitus. | lld:pubmed |
| pubmed-article:18725054 | pubmed:affiliation | Second Year Medical Students, Dubai Medical College for Girls, Dubai, UAE. | lld:pubmed |
| pubmed-article:18725054 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18725054 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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