18648532

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pubmed-article:18648532	lifeskim:mentions	umls-concept:C0002736	lld:lifeskim
pubmed-article:18648532	lifeskim:mentions	umls-concept:C0026809	lld:lifeskim
pubmed-article:18648532	lifeskim:mentions	umls-concept:C0598958	lld:lifeskim
pubmed-article:18648532	lifeskim:mentions	umls-concept:C0439662	lld:lifeskim
pubmed-article:18648532	pubmed:issue	7	lld:pubmed
pubmed-article:18648532	pubmed:dateCreated	2008-7-23	lld:pubmed
pubmed-article:18648532	pubmed:abstractText	Innate neuroimmune dysfunction is a pathobiological feature of amyotrophic lateral sclerosis (ALS). However, links, if any, between disease and adaptive immunity are poorly understood. Thus, the role of T cell immunity in disease was investigated in human G93A superoxide dismutase 1 (SOD1) transgenic (Tg) mice and subsequently in ALS patients.	lld:pubmed
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