pubmed-article:18648526 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:18648526 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
pubmed-article:18648526 | lifeskim:mentions | umls-concept:C0010453 | lld:lifeskim |
pubmed-article:18648526 | lifeskim:mentions | umls-concept:C2325187 | lld:lifeskim |
pubmed-article:18648526 | lifeskim:mentions | umls-concept:C0011777 | lld:lifeskim |
pubmed-article:18648526 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
pubmed-article:18648526 | pubmed:dateCreated | 2008-7-23 | lld:pubmed |
pubmed-article:18648526 | pubmed:abstractText | Treatment with glucocorticoids is a well known risk factor for cataract development, although the pathogenic mechanism has not been elucidated. The aim of the study was to investigate the effects of glucocorticoids in cultured human lens epithelial cells. | lld:pubmed |
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pubmed-article:18648526 | pubmed:language | eng | lld:pubmed |
pubmed-article:18648526 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18648526 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:18648526 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:18648526 | pubmed:issn | 1090-0535 | lld:pubmed |
pubmed-article:18648526 | pubmed:author | pubmed-author:PetersenAA | lld:pubmed |
pubmed-article:18648526 | pubmed:author | pubmed-author:CarlssonTT | lld:pubmed |
pubmed-article:18648526 | pubmed:author | pubmed-author:ZetterbergMM | lld:pubmed |
pubmed-article:18648526 | pubmed:author | pubmed-author:KarlssonJ-OJO | lld:pubmed |
pubmed-article:18648526 | pubmed:author | pubmed-author:JonhedeSS | lld:pubmed |
pubmed-article:18648526 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:18648526 | pubmed:volume | 14 | lld:pubmed |
pubmed-article:18648526 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:18648526 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:18648526 | pubmed:pagination | 1344-52 | lld:pubmed |
pubmed-article:18648526 | pubmed:dateRevised | 2010-9-22 | lld:pubmed |
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