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pubmed-article:18590437pubmed:abstractTextThe authors previously demonstrated that 17beta-estradiol benzoate (E2) treatment prevents subarachnoid hemorrhage (SAH)-induced cerebral vasospasm and preserves endothelial nitric oxide synthase (eNOS) in male rats. Changes in the expression of estrogen receptor (ER) subtypes ERalpha and -beta and their roles in the E2-mediated preservation of eNOS in SAH remain unknown. In the present study the effects of SAH on the expression of ERalpha and -beta in the cerebral arteries were clarified, and the receptor roles in the E2-mediated preservation of eNOS expression in SAH were differentiated.lld:pubmed
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pubmed-article:18590437pubmed:authorpubmed-author:LinChih-LungC...lld:pubmed
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pubmed-article:18590437pubmed:dateRevised2009-11-19lld:pubmed
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pubmed-article:18590437pubmed:articleTitleUpregulation of estrogen receptor alpha and mediation of 17beta-estradiol vasoprotective effects via estrogen receptor alpha in basilar arteries in rats after experimental subarachnoid hemorrhage.lld:pubmed
pubmed-article:18590437pubmed:affiliationGraduate Institute of Medicine, College of Medicine, Kaohsiung Medical University Hospital, Kaohsiung, Taiwan.lld:pubmed
pubmed-article:18590437pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:18590437pubmed:publicationTypeResearch Support, Non-U.S. Gov'tlld:pubmed