pubmed-article:18559660 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:18559660 | lifeskim:mentions | umls-concept:C0011854 | lld:lifeskim |
pubmed-article:18559660 | lifeskim:mentions | umls-concept:C0919453 | lld:lifeskim |
pubmed-article:18559660 | lifeskim:mentions | umls-concept:C0011881 | lld:lifeskim |
pubmed-article:18559660 | lifeskim:mentions | umls-concept:C0023745 | lld:lifeskim |
pubmed-article:18559660 | lifeskim:mentions | umls-concept:C0752046 | lld:lifeskim |
pubmed-article:18559660 | lifeskim:mentions | umls-concept:C0034606 | lld:lifeskim |
pubmed-article:18559660 | lifeskim:mentions | umls-concept:C0441633 | lld:lifeskim |
pubmed-article:18559660 | lifeskim:mentions | umls-concept:C0449445 | lld:lifeskim |
pubmed-article:18559660 | pubmed:issue | 9 | lld:pubmed |
pubmed-article:18559660 | pubmed:dateCreated | 2008-8-28 | lld:pubmed |
pubmed-article:18559660 | pubmed:abstractText | Epidemiological and family studies have demonstrated that susceptibility genes play an important role in the etiology of diabetic nephropathy, defined as persistent proteinuria or end-stage renal disease (ESRD) in type 1 diabetes. | lld:pubmed |
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pubmed-article:18559660 | pubmed:language | eng | lld:pubmed |
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pubmed-article:18559660 | pubmed:citationSubset | AIM | lld:pubmed |
pubmed-article:18559660 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:18559660 | pubmed:month | Sep | lld:pubmed |
pubmed-article:18559660 | pubmed:issn | 1939-327X | lld:pubmed |
pubmed-article:18559660 | pubmed:author | pubmed-author:RogusJohn JJJ | lld:pubmed |
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pubmed-article:18559660 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:18559660 | pubmed:volume | 57 | lld:pubmed |
pubmed-article:18559660 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:18559660 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:18559660 | pubmed:pagination | 2519-26 | lld:pubmed |
pubmed-article:18559660 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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pubmed-article:18559660 | pubmed:year | 2008 | lld:pubmed |
pubmed-article:18559660 | pubmed:articleTitle | High-density single nucleotide polymorphism genome-wide linkage scan for susceptibility genes for diabetic nephropathy in type 1 diabetes: discordant sibpair approach. | lld:pubmed |
pubmed-article:18559660 | pubmed:affiliation | Research Division, Joslin Diabetes Center, Boston, Massachusetts, USA. | lld:pubmed |
pubmed-article:18559660 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:18559660 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
pubmed-article:18559660 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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