| pubmed-article:18509093 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18509093 | lifeskim:mentions | umls-concept:C0009247 | lld:lifeskim |
| pubmed-article:18509093 | lifeskim:mentions | umls-concept:C0002395 | lld:lifeskim |
| pubmed-article:18509093 | lifeskim:mentions | umls-concept:C0003211 | lld:lifeskim |
| pubmed-article:18509093 | lifeskim:mentions | umls-concept:C1708528 | lld:lifeskim |
| pubmed-article:18509093 | lifeskim:mentions | umls-concept:C0308269 | lld:lifeskim |
| pubmed-article:18509093 | lifeskim:mentions | umls-concept:C2349200 | lld:lifeskim |
| pubmed-article:18509093 | lifeskim:mentions | umls-concept:C1709595 | lld:lifeskim |
| pubmed-article:18509093 | lifeskim:mentions | umls-concept:C0758515 | lld:lifeskim |
| pubmed-article:18509093 | pubmed:issue | 24 | lld:pubmed |
| pubmed-article:18509093 | pubmed:dateCreated | 2008-6-10 | lld:pubmed |
| pubmed-article:18509093 | pubmed:abstractText | Observational studies show reduced incidence of Alzheimer dementia (AD) in users of nonsteroidal anti-inflammatory drugs (NSAIDs). One hypothesis holds that the subset of NSAIDs known as selective A beta(42)-lowering agents (SALAs) is responsible for this apparent reduction in AD risk. | lld:pubmed |
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