pubmed-article:18477810 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:18477810 | lifeskim:mentions | umls-concept:C0085243 | lld:lifeskim |
pubmed-article:18477810 | lifeskim:mentions | umls-concept:C0019796 | lld:lifeskim |
pubmed-article:18477810 | lifeskim:mentions | umls-concept:C0521119 | lld:lifeskim |
pubmed-article:18477810 | lifeskim:mentions | umls-concept:C0005525 | lld:lifeskim |
pubmed-article:18477810 | lifeskim:mentions | umls-concept:C0011847 | lld:lifeskim |
pubmed-article:18477810 | lifeskim:mentions | umls-concept:C0443146 | lld:lifeskim |
pubmed-article:18477810 | lifeskim:mentions | umls-concept:C0449258 | lld:lifeskim |
pubmed-article:18477810 | lifeskim:mentions | umls-concept:C1333908 | lld:lifeskim |
pubmed-article:18477810 | lifeskim:mentions | umls-concept:C1551336 | lld:lifeskim |
pubmed-article:18477810 | lifeskim:mentions | umls-concept:C2349975 | lld:lifeskim |
pubmed-article:18477810 | lifeskim:mentions | umls-concept:C0332162 | lld:lifeskim |
pubmed-article:18477810 | pubmed:issue | 8 | lld:pubmed |
pubmed-article:18477810 | pubmed:dateCreated | 2008-7-29 | lld:pubmed |
pubmed-article:18477810 | pubmed:abstractText | The implication of innate immunity in type 1 diabetes development has long been proposed. High-mobility group box 1 (HMGB1), an evolutionarily conserved chromosomal protein, was recently recognized to be a potent innate inflammatory mediator when released extracellularly. We sought to test the hypothesis that HMGB1 acts as an innate immune mediator implicated in type 1 diabetes pathogenesis. | lld:pubmed |
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pubmed-article:18477810 | pubmed:language | eng | lld:pubmed |
pubmed-article:18477810 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18477810 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:18477810 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:18477810 | pubmed:month | Aug | lld:pubmed |
pubmed-article:18477810 | pubmed:issn | 1939-327X | lld:pubmed |
pubmed-article:18477810 | pubmed:author | pubmed-author:HuangYafeiY | lld:pubmed |
pubmed-article:18477810 | pubmed:author | pubmed-author:WangYingY | lld:pubmed |
pubmed-article:18477810 | pubmed:author | pubmed-author:WangMong-Heng... | lld:pubmed |
pubmed-article:18477810 | pubmed:author | pubmed-author:YangPingP | lld:pubmed |
pubmed-article:18477810 | pubmed:author | pubmed-author:SheJin-XiongJ... | lld:pubmed |
pubmed-article:18477810 | pubmed:author | pubmed-author:SternDavid... | lld:pubmed |
pubmed-article:18477810 | pubmed:author | pubmed-author:DongZhengZ | lld:pubmed |
pubmed-article:18477810 | pubmed:author | pubmed-author:GongFeiliF | lld:pubmed |
pubmed-article:18477810 | pubmed:author | pubmed-author:WangCong-YiCY | lld:pubmed |
pubmed-article:18477810 | pubmed:author | pubmed-author:WeiWenzhongW | lld:pubmed |
pubmed-article:18477810 | pubmed:author | pubmed-author:PurohitSharad... | lld:pubmed |
pubmed-article:18477810 | pubmed:author | pubmed-author:HanJunyanJ | lld:pubmed |
pubmed-article:18477810 | pubmed:author | pubmed-author:ZhongJinxinJ | lld:pubmed |
pubmed-article:18477810 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:18477810 | pubmed:volume | 57 | lld:pubmed |
pubmed-article:18477810 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:18477810 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:18477810 | pubmed:pagination | 2118-27 | lld:pubmed |
pubmed-article:18477810 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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