| pubmed-article:18452260 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18452260 | lifeskim:mentions | umls-concept:C0030705 | lld:lifeskim |
| pubmed-article:18452260 | lifeskim:mentions | umls-concept:C1956346 | lld:lifeskim |
| pubmed-article:18452260 | lifeskim:mentions | umls-concept:C0033706 | lld:lifeskim |
| pubmed-article:18452260 | lifeskim:mentions | umls-concept:C1521761 | lld:lifeskim |
| pubmed-article:18452260 | lifeskim:mentions | umls-concept:C0678951 | lld:lifeskim |
| pubmed-article:18452260 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:18452260 | pubmed:dateCreated | 2008-5-2 | lld:pubmed |
| pubmed-article:18452260 | pubmed:abstractText | The precise molecular mechanisms culminating in coronary artery disease (CAD) are not well understood, despite a wealth of knowledge on predisposing risk factors and pathomechanisms. CAD and myocardial infarction (MI) are complex genetic diseases; neither the environment alone, nor a single gene, cause disease, rather, a mix of environmental and genetic factors lead to atherosclerosis of the coronary arteries. | lld:pubmed |
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| pubmed-article:18452260 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:18452260 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:18452260 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18452260 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:18452260 | pubmed:issn | 0513-5796 | lld:pubmed |
| pubmed-article:18452260 | pubmed:author | pubmed-author:TamerLülüferL | lld:pubmed |
| pubmed-article:18452260 | pubmed:author | pubmed-author:SucuNehirN | lld:pubmed |
| pubmed-article:18452260 | pubmed:author | pubmed-author:ErcanBahadirB | lld:pubmed |
| pubmed-article:18452260 | pubmed:author | pubmed-author:AtikU?urU | lld:pubmed |
| pubmed-article:18452260 | pubmed:author | pubmed-author:CamsariAhmetA | lld:pubmed |
| pubmed-article:18452260 | pubmed:author | pubmed-author:PekdemirHasan... | lld:pubmed |
| pubmed-article:18452260 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:18452260 | pubmed:day | 30 | lld:pubmed |
| pubmed-article:18452260 | pubmed:volume | 49 | lld:pubmed |
| pubmed-article:18452260 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18452260 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18452260 | pubmed:pagination | 237-43 | lld:pubmed |
| pubmed-article:18452260 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:18452260 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18452260 | pubmed:articleTitle | Factor VLeiden and prothrombin G20210A gene polymorphisms in patients with coronary artery disease. | lld:pubmed |
| pubmed-article:18452260 | pubmed:affiliation | Department of Biochemistry, Mersin University, Medical Faculty, Mersin, Turkey. | lld:pubmed |
| pubmed-article:18452260 | pubmed:publicationType | Journal Article | lld:pubmed |
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