pubmed-article:18417658 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:18417658 | lifeskim:mentions | umls-concept:C0020792 | lld:lifeskim |
pubmed-article:18417658 | lifeskim:mentions | umls-concept:C0038172 | lld:lifeskim |
pubmed-article:18417658 | lifeskim:mentions | umls-concept:C0600688 | lld:lifeskim |
pubmed-article:18417658 | lifeskim:mentions | umls-concept:C0370215 | lld:lifeskim |
pubmed-article:18417658 | lifeskim:mentions | umls-concept:C1521761 | lld:lifeskim |
pubmed-article:18417658 | lifeskim:mentions | umls-concept:C0205210 | lld:lifeskim |
pubmed-article:18417658 | lifeskim:mentions | umls-concept:C1880177 | lld:lifeskim |
pubmed-article:18417658 | pubmed:issue | 6 | lld:pubmed |
pubmed-article:18417658 | pubmed:dateCreated | 2008-6-9 | lld:pubmed |
pubmed-article:18417658 | pubmed:abstractText | We examined the ability of 206 clinical isolates of Staphylococcus aureus to lyse T cells and found differences between Agr groups. We found that the beta and delta hemolysins are involved and that methicillin-resistant S. aureus strains are less toxic than methicillin-susceptible S. aureus strains. | lld:pubmed |
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pubmed-article:18417658 | pubmed:language | eng | lld:pubmed |
pubmed-article:18417658 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18417658 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:18417658 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18417658 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18417658 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:18417658 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:18417658 | pubmed:month | Jun | lld:pubmed |
pubmed-article:18417658 | pubmed:issn | 1098-660X | lld:pubmed |
pubmed-article:18417658 | pubmed:author | pubmed-author:BucklingAngus... | lld:pubmed |
pubmed-article:18417658 | pubmed:author | pubmed-author:MasseyRuth... | lld:pubmed |
pubmed-article:18417658 | pubmed:author | pubmed-author:CollinsJamesJ | lld:pubmed |
pubmed-article:18417658 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:18417658 | pubmed:volume | 46 | lld:pubmed |
pubmed-article:18417658 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:18417658 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:18417658 | pubmed:pagination | 2112-4 | lld:pubmed |
pubmed-article:18417658 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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pubmed-article:18417658 | pubmed:meshHeading | pubmed-meshheading:18417658... | lld:pubmed |
pubmed-article:18417658 | pubmed:year | 2008 | lld:pubmed |
pubmed-article:18417658 | pubmed:articleTitle | Identification of factors contributing to T-cell toxicity of Staphylococcus aureus clinical isolates. | lld:pubmed |
pubmed-article:18417658 | pubmed:affiliation | Department of Zoology, University of Oxford, Oxford OX1 3PS, United Kingdom. | lld:pubmed |
pubmed-article:18417658 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:18417658 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |