pubmed-article:18405362 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:18405362 | lifeskim:mentions | umls-concept:C0006826 | lld:lifeskim |
pubmed-article:18405362 | lifeskim:mentions | umls-concept:C0205145 | lld:lifeskim |
pubmed-article:18405362 | lifeskim:mentions | umls-concept:C0027627 | lld:lifeskim |
pubmed-article:18405362 | lifeskim:mentions | umls-concept:C2745955 | lld:lifeskim |
pubmed-article:18405362 | lifeskim:mentions | umls-concept:C0439064 | lld:lifeskim |
pubmed-article:18405362 | pubmed:dateCreated | 2008-5-8 | lld:pubmed |
pubmed-article:18405362 | pubmed:abstractText | Occurrence of tumors at multiple sites is a hallmark of malignant cancers and contributes to the high mortality of cancers. The formation of multi-site cancers (MSCs) has conventionally been regarded as a result of hematogenous metastasis. However, some MSCs may appear as unusual in the sense of vascular dissemination pattern and therefore be explained by alternative metastasis models or even by non-metastatic independent formation mechanisms. | lld:pubmed |
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