| pubmed-article:18395080 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18395080 | lifeskim:mentions | umls-concept:C0030705 | lld:lifeskim |
| pubmed-article:18395080 | lifeskim:mentions | umls-concept:C1333990 | lld:lifeskim |
| pubmed-article:18395080 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
| pubmed-article:18395080 | lifeskim:mentions | umls-concept:C0301872 | lld:lifeskim |
| pubmed-article:18395080 | lifeskim:mentions | umls-concept:C1742737 | lld:lifeskim |
| pubmed-article:18395080 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:18395080 | pubmed:dateCreated | 2008-4-8 | lld:pubmed |
| pubmed-article:18395080 | pubmed:abstractText | Colorectal cancers (CRC) associated with the hereditary nonpolyposis colorectal cancer (HNPCC) syndrome display high-level microsatellite instability (MSI-H) as a consequence of mismatch repair deficiency. HNPCC-associated CRC frequently show features of a pronounced immune response, most likely resulting from the MSI-induced generation of novel tumor-specific carboxy-terminal frameshift peptides (FSPs). However, the role of FSP-specific immune surveillance mechanisms in HNPCC are unknown at present. | lld:pubmed |
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| pubmed-article:18395080 | pubmed:language | eng | lld:pubmed |
| pubmed-article:18395080 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18395080 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:18395080 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18395080 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:18395080 | pubmed:issn | 1528-0012 | lld:pubmed |
| pubmed-article:18395080 | pubmed:author | pubmed-author:BennerAxelA | lld:pubmed |
| pubmed-article:18395080 | pubmed:author | pubmed-author:von Knebel... | lld:pubmed |
| pubmed-article:18395080 | pubmed:author | pubmed-author:LinnebacherMi... | lld:pubmed |
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| pubmed-article:18395080 | pubmed:author | pubmed-author:KienlePeterP | lld:pubmed |
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| pubmed-article:18395080 | pubmed:author | pubmed-author:TariverdianMi... | lld:pubmed |
| pubmed-article:18395080 | pubmed:author | pubmed-author:EiermannSusan... | lld:pubmed |
| pubmed-article:18395080 | pubmed:author | pubmed-author:KnaebelHanns... | lld:pubmed |
| pubmed-article:18395080 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18395080 | pubmed:volume | 134 | lld:pubmed |
| pubmed-article:18395080 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18395080 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18395080 | pubmed:pagination | 988-97 | lld:pubmed |
| pubmed-article:18395080 | pubmed:dateRevised | 2008-9-2 | lld:pubmed |
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| pubmed-article:18395080 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18395080 | pubmed:articleTitle | Immune response against frameshift-induced neopeptides in HNPCC patients and healthy HNPCC mutation carriers. | lld:pubmed |
| pubmed-article:18395080 | pubmed:affiliation | Department of Applied Tumor Biology, Institute of Pathology, University of Heidelberg, and Group Cancer Early Detection, German Cancer Research Center (DKFZ), Molecular Medicine Partnership Unit (MMPU), Heidelberg, Germany. | lld:pubmed |
| pubmed-article:18395080 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18395080 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:18395080 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:18395080 | pubmed:publicationType | Multicenter Study | lld:pubmed |
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