| pubmed-article:18394191 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18394191 | lifeskim:mentions | umls-concept:C0034802 | lld:lifeskim |
| pubmed-article:18394191 | lifeskim:mentions | umls-concept:C0014139 | lld:lifeskim |
| pubmed-article:18394191 | lifeskim:mentions | umls-concept:C1947906 | lld:lifeskim |
| pubmed-article:18394191 | lifeskim:mentions | umls-concept:C0441712 | lld:lifeskim |
| pubmed-article:18394191 | pubmed:dateCreated | 2008-5-13 | lld:pubmed |
| pubmed-article:18394191 | pubmed:abstractText | The Epidermal Growth Factor (EGF) receptor has been shown to internalize via clathrin-independent endocytosis (CIE) in a ligand concentration dependent manner. From a modeling point of view, this resembles an ultrasensitive response, which is the ability of signaling networks to suppress a response for low input values and to increase to a pre-defined level for inputs exceeding a certain threshold. Several mechanisms to generate this behaviour have been described theoretically, the underlying assumptions of which, however, have not been experimentally demonstrated for the EGF receptor internalization network. | lld:pubmed |
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| pubmed-article:18394191 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:18394191 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:18394191 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18394191 | pubmed:issn | 1752-0509 | lld:pubmed |
| pubmed-article:18394191 | pubmed:author | pubmed-author:EilsRolandR | lld:pubmed |
| pubmed-article:18394191 | pubmed:author | pubmed-author:DikicIvanI | lld:pubmed |
| pubmed-article:18394191 | pubmed:author | pubmed-author:HoellerDaniel... | lld:pubmed |
| pubmed-article:18394191 | pubmed:author | pubmed-author:Schmidt-Glene... | lld:pubmed |
| pubmed-article:18394191 | pubmed:author | pubmed-author:VachevaIvayla... | lld:pubmed |
| pubmed-article:18394191 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18394191 | pubmed:volume | 2 | lld:pubmed |
| pubmed-article:18394191 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18394191 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18394191 | pubmed:pagination | 32 | lld:pubmed |
| pubmed-article:18394191 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:18394191 | pubmed:meshHeading | pubmed-meshheading:18394191... | lld:pubmed |
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| pubmed-article:18394191 | pubmed:meshHeading | pubmed-meshheading:18394191... | lld:pubmed |
| pubmed-article:18394191 | pubmed:meshHeading | pubmed-meshheading:18394191... | lld:pubmed |
| pubmed-article:18394191 | pubmed:meshHeading | pubmed-meshheading:18394191... | lld:pubmed |
| pubmed-article:18394191 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18394191 | pubmed:articleTitle | An ultrasensitive sorting mechanism for EGF receptor endocytosis. | lld:pubmed |
| pubmed-article:18394191 | pubmed:affiliation | Division Theoretical Bioinformatics, German Cancer Research Center (DKFZ), 69120 Heidelberg, Germany. H.Schmidt-Glenewinkel@dkfz.de | lld:pubmed |
| pubmed-article:18394191 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18394191 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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