| pubmed-article:18325124 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18325124 | lifeskim:mentions | umls-concept:C0008633 | lld:lifeskim |
| pubmed-article:18325124 | lifeskim:mentions | umls-concept:C0032150 | lld:lifeskim |
| pubmed-article:18325124 | lifeskim:mentions | umls-concept:C2717927 | lld:lifeskim |
| pubmed-article:18325124 | lifeskim:mentions | umls-concept:C0205250 | lld:lifeskim |
| pubmed-article:18325124 | lifeskim:mentions | umls-concept:C1548673 | lld:lifeskim |
| pubmed-article:18325124 | lifeskim:mentions | umls-concept:C1707513 | lld:lifeskim |
| pubmed-article:18325124 | pubmed:dateCreated | 2008-4-3 | lld:pubmed |
| pubmed-article:18325124 | pubmed:abstractText | Segmental duplications (SD) have been found in genomes of various organisms, often accumulated at the ends of chromosomes. It has been assumed that the sequence homology in-between the SDs allow for ectopic interactions that may contribute to the emergence of new genes or gene variants through recombinatorial events. | lld:pubmed |
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| pubmed-article:18325124 | pubmed:language | eng | lld:pubmed |
| pubmed-article:18325124 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18325124 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:18325124 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18325124 | pubmed:issn | 1475-2875 | lld:pubmed |
| pubmed-article:18325124 | pubmed:author | pubmed-author:WahlgrenMatsM | lld:pubmed |
| pubmed-article:18325124 | pubmed:author | pubmed-author:MokBobo WBW | lld:pubmed |
| pubmed-article:18325124 | pubmed:author | pubmed-author:RibackeUlfU | lld:pubmed |
| pubmed-article:18325124 | pubmed:author | pubmed-author:SherwoodEllen... | lld:pubmed |
| pubmed-article:18325124 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18325124 | pubmed:volume | 7 | lld:pubmed |
| pubmed-article:18325124 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18325124 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18325124 | pubmed:pagination | 46 | lld:pubmed |
| pubmed-article:18325124 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
| pubmed-article:18325124 | pubmed:meshHeading | pubmed-meshheading:18325124... | lld:pubmed |
| pubmed-article:18325124 | pubmed:meshHeading | pubmed-meshheading:18325124... | lld:pubmed |
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| pubmed-article:18325124 | pubmed:meshHeading | pubmed-meshheading:18325124... | lld:pubmed |
| pubmed-article:18325124 | pubmed:meshHeading | pubmed-meshheading:18325124... | lld:pubmed |
| pubmed-article:18325124 | pubmed:meshHeading | pubmed-meshheading:18325124... | lld:pubmed |
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| pubmed-article:18325124 | pubmed:meshHeading | pubmed-meshheading:18325124... | lld:pubmed |
| pubmed-article:18325124 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18325124 | pubmed:articleTitle | A highly conserved segmental duplication in the subtelomeres of Plasmodium falciparum chromosomes varies in copy number. | lld:pubmed |
| pubmed-article:18325124 | pubmed:affiliation | Department of Microbiology, Tumor and Cell Biology (MTC), Karolinska Institutet, SE-171 77 Stockholm, Sweden. bobo.mok@ki.se | lld:pubmed |
| pubmed-article:18325124 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18325124 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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