In hyperthyroidism, tissue glucose disposal is increased to adapt to high energy demand. Our aim was to examine the regulation of glucose transporter (GLUT) isoforms by IGF-I in monocytes from patients with hyperthyroidism.
Second Department of Internal Medicine, Research Institute and Diabetes Center, University General Hospital Attikon, Athens University, 1 Rimini Street, 12462 Haidari, Greece. gdimi@ath.forthnet.gr
