pubmed-article:18288727 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:18288727 | lifeskim:mentions | umls-concept:C0012356 | lld:lifeskim |
pubmed-article:18288727 | lifeskim:mentions | umls-concept:C0221464 | lld:lifeskim |
pubmed-article:18288727 | lifeskim:mentions | umls-concept:C1522318 | lld:lifeskim |
pubmed-article:18288727 | lifeskim:mentions | umls-concept:C0012359 | lld:lifeskim |
pubmed-article:18288727 | lifeskim:mentions | umls-concept:C1948041 | lld:lifeskim |
pubmed-article:18288727 | lifeskim:mentions | umls-concept:C1521761 | lld:lifeskim |
pubmed-article:18288727 | lifeskim:mentions | umls-concept:C0522523 | lld:lifeskim |
pubmed-article:18288727 | pubmed:issue | 3 | lld:pubmed |
pubmed-article:18288727 | pubmed:dateCreated | 2008-2-26 | lld:pubmed |
pubmed-article:18288727 | pubmed:abstractText | It has previously been observed that coronary diameter may increase following relief of flow-limiting obstruction. Flow mediated dilatation (FMD) is a fundamental adaptive mechanism for arteries, which is dependent on intact endothelial function. We thus aimed to characterize whether the degree of this flow-mediated dilatation was related to risk factors, which may impair endothelial function. | lld:pubmed |
pubmed-article:18288727 | pubmed:language | eng | lld:pubmed |
pubmed-article:18288727 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18288727 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:18288727 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:18288727 | pubmed:month | Feb | lld:pubmed |
pubmed-article:18288727 | pubmed:issn | 1522-726X | lld:pubmed |
pubmed-article:18288727 | pubmed:author | pubmed-author:CelermajerDav... | lld:pubmed |
pubmed-article:18288727 | pubmed:author | pubmed-author:AdamsMark RMR | lld:pubmed |
pubmed-article:18288727 | pubmed:author | pubmed-author:ThanyasiriPan... | lld:pubmed |
pubmed-article:18288727 | pubmed:author | pubmed-author:McGradyMichel... | lld:pubmed |
pubmed-article:18288727 | pubmed:author | pubmed-author:BaileyBrian... | lld:pubmed |
pubmed-article:18288727 | pubmed:copyrightInfo | (c) 2008 Wiley-Liss, Inc. | lld:pubmed |
pubmed-article:18288727 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:18288727 | pubmed:day | 15 | lld:pubmed |
pubmed-article:18288727 | pubmed:volume | 71 | lld:pubmed |
pubmed-article:18288727 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:18288727 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:18288727 | pubmed:pagination | 327-32 | lld:pubmed |
pubmed-article:18288727 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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pubmed-article:18288727 | pubmed:year | 2008 | lld:pubmed |
pubmed-article:18288727 | pubmed:articleTitle | Determinants of coronary arterial flow-mediated dilatation following percutaneous coronary intervention. | lld:pubmed |
pubmed-article:18288727 | pubmed:affiliation | Department of Cardiology at Royal Prince Alfred Hospital, Sydney, Australia. | lld:pubmed |
pubmed-article:18288727 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:18288727 | pubmed:publicationType | Comparative Study | lld:pubmed |
pubmed-article:18288727 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |