| pubmed-article:18270339 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18270339 | lifeskim:mentions | umls-concept:C0035647 | lld:lifeskim |
| pubmed-article:18270339 | lifeskim:mentions | umls-concept:C0025286 | lld:lifeskim |
| pubmed-article:18270339 | lifeskim:mentions | umls-concept:C0205419 | lld:lifeskim |
| pubmed-article:18270339 | lifeskim:mentions | umls-concept:C1333237 | lld:lifeskim |
| pubmed-article:18270339 | lifeskim:mentions | umls-concept:C0936012 | lld:lifeskim |
| pubmed-article:18270339 | lifeskim:mentions | umls-concept:C1880156 | lld:lifeskim |
| pubmed-article:18270339 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:18270339 | pubmed:dateCreated | 2008-2-20 | lld:pubmed |
| pubmed-article:18270339 | pubmed:abstractText | Meningiomas account for up to 37% of all primary brain tumors. Genetic susceptibility to meningioma is well established, with the risk among relatives of meningioma patients being approximately threefold higher than that in the general population. A relationship between risk of meningioma and exposure to ionizing radiation is also well known and led us to examine whether variants in DNA repair genes contribute to disease susceptibility. | lld:pubmed |
| pubmed-article:18270339 | pubmed:language | eng | lld:pubmed |
| pubmed-article:18270339 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18270339 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:18270339 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18270339 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:18270339 | pubmed:issn | 1460-2105 | lld:pubmed |
| pubmed-article:18270339 | pubmed:author | pubmed-author:KosteljanetzM... | lld:pubmed |
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| pubmed-article:18270339 | pubmed:author | pubmed-author:FeychtingMari... | lld:pubmed |
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| pubmed-article:18270339 | pubmed:author | pubmed-author:AhlbomAndersA | lld:pubmed |
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| pubmed-article:18270339 | pubmed:author | pubmed-author:ChristensenHe... | lld:pubmed |
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| pubmed-article:18270339 | pubmed:author | pubmed-author:LophatananonA... | lld:pubmed |
| pubmed-article:18270339 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18270339 | pubmed:day | 20 | lld:pubmed |
| pubmed-article:18270339 | pubmed:volume | 100 | lld:pubmed |
| pubmed-article:18270339 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18270339 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18270339 | pubmed:pagination | 270-6 | lld:pubmed |
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| pubmed-article:18270339 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18270339 | pubmed:articleTitle | Comprehensive analysis of DNA repair gene variants and risk of meningioma. | lld:pubmed |
| pubmed-article:18270339 | pubmed:affiliation | Section of Cancer Genetics, Institute of Cancer Research, 15 Cotswold Rd, Sutton, Surrey SM2 5NG, UK. | lld:pubmed |
| pubmed-article:18270339 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18270339 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:18270339 | pubmed:publicationType | Multicenter Study | lld:pubmed |
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