| pubmed-article:18221926 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18221926 | lifeskim:mentions | umls-concept:C0032961 | lld:lifeskim |
| pubmed-article:18221926 | lifeskim:mentions | umls-concept:C0032914 | lld:lifeskim |
| pubmed-article:18221926 | lifeskim:mentions | umls-concept:C1708096 | lld:lifeskim |
| pubmed-article:18221926 | lifeskim:mentions | umls-concept:C2698872 | lld:lifeskim |
| pubmed-article:18221926 | lifeskim:mentions | umls-concept:C0009546 | lld:lifeskim |
| pubmed-article:18221926 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
| pubmed-article:18221926 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:18221926 | pubmed:dateCreated | 2008-4-8 | lld:pubmed |
| pubmed-article:18221926 | pubmed:abstractText | Preeclampsia is a multisystem disease classically defined on the basis of hypertension and proteinuria. As shown in animal studies, complement activation is associated with inflammation in the placenta and adverse pregnancy outcomes. The association between complement activation in humans and adverse pregnancy outcomes is unclear. The purpose of this study was to determine whether elevated levels of the activation fragment Bb in early pregnancy are predictive of preeclampsia. | lld:pubmed |
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| pubmed-article:18221926 | pubmed:language | eng | lld:pubmed |
| pubmed-article:18221926 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18221926 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:18221926 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18221926 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:18221926 | pubmed:issn | 1097-6868 | lld:pubmed |
| pubmed-article:18221926 | pubmed:author | pubmed-author:EssawiG HGH | lld:pubmed |
| pubmed-article:18221926 | pubmed:author | pubmed-author:MontefalcoP... | lld:pubmed |
| pubmed-article:18221926 | pubmed:author | pubmed-author:HolersV... | lld:pubmed |
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| pubmed-article:18221926 | pubmed:author | pubmed-author:ByersTimT | lld:pubmed |
| pubmed-article:18221926 | pubmed:author | pubmed-author:NevilleMargar... | lld:pubmed |
| pubmed-article:18221926 | pubmed:author | pubmed-author:LynchAnne MAM | lld:pubmed |
| pubmed-article:18221926 | pubmed:author | pubmed-author:GiclasPatrici... | lld:pubmed |
| pubmed-article:18221926 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18221926 | pubmed:volume | 198 | lld:pubmed |
| pubmed-article:18221926 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18221926 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18221926 | pubmed:pagination | 385.e1-9 | lld:pubmed |
| pubmed-article:18221926 | pubmed:dateRevised | 2011-8-1 | lld:pubmed |
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| pubmed-article:18221926 | pubmed:meshHeading | pubmed-meshheading:18221926... | lld:pubmed |
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| pubmed-article:18221926 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18221926 | pubmed:articleTitle | Alternative complement pathway activation fragment Bb in early pregnancy as a predictor of preeclampsia. | lld:pubmed |
| pubmed-article:18221926 | pubmed:affiliation | Department of Obstetrics and Gynecology, University of Colorado at Denver and Health Sciences Center, Denver, CO, USA. anne.lynch@uchsc.edu | lld:pubmed |
| pubmed-article:18221926 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18221926 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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