| pubmed-article:18205950 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18205950 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:18205950 | lifeskim:mentions | umls-concept:C0678222 | lld:lifeskim |
| pubmed-article:18205950 | lifeskim:mentions | umls-concept:C0334227 | lld:lifeskim |
| pubmed-article:18205950 | lifeskim:mentions | umls-concept:C1514559 | lld:lifeskim |
| pubmed-article:18205950 | lifeskim:mentions | umls-concept:C1539270 | lld:lifeskim |
| pubmed-article:18205950 | lifeskim:mentions | umls-concept:C2610698 | lld:lifeskim |
| pubmed-article:18205950 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:18205950 | pubmed:dateCreated | 2008-4-25 | lld:pubmed |
| pubmed-article:18205950 | pubmed:abstractText | Aberrant microenvironment and endoplasmic reticulum (ER) stress are associated with solid-tumor progression. Stress proteins, like heat shock proteins and glucose-regulated proteins, are frequently overexpressed in human tumors. It has been reported that derlin-1 is involved in ER stress response. In vitro studies have demonstrated that derlin-1 participates in the retrotranslocation of misfolded proteins from ER into the cytosol. Because the roles of derlin-1 in human cancer have not yet been characterized, we investigated the expression of derlin-1 in human breast carcinoma and whether it protected cancer cells against ER stress-induced apoptosis. | lld:pubmed |
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| pubmed-article:18205950 | pubmed:language | eng | lld:pubmed |
| pubmed-article:18205950 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18205950 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:18205950 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18205950 | pubmed:issn | 1465-542X | lld:pubmed |
| pubmed-article:18205950 | pubmed:author | pubmed-author:LDD | lld:pubmed |
| pubmed-article:18205950 | pubmed:author | pubmed-author:JiangYangfuY | lld:pubmed |
| pubmed-article:18205950 | pubmed:author | pubmed-author:LiuWeipingW | lld:pubmed |
| pubmed-article:18205950 | pubmed:author | pubmed-author:RanYuliangY | lld:pubmed |
| pubmed-article:18205950 | pubmed:author | pubmed-author:YangZhihuaZ | lld:pubmed |
| pubmed-article:18205950 | pubmed:author | pubmed-author:WangJiaoJ | lld:pubmed |
| pubmed-article:18205950 | pubmed:author | pubmed-author:ZhangHongyinH | lld:pubmed |
| pubmed-article:18205950 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18205950 | pubmed:volume | 10 | lld:pubmed |
| pubmed-article:18205950 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18205950 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18205950 | pubmed:pagination | R7 | lld:pubmed |
| pubmed-article:18205950 | pubmed:dateRevised | 2010-3-23 | lld:pubmed |
| pubmed-article:18205950 | pubmed:meshHeading | pubmed-meshheading:18205950... | lld:pubmed |
| pubmed-article:18205950 | pubmed:meshHeading | pubmed-meshheading:18205950... | lld:pubmed |
| pubmed-article:18205950 | pubmed:meshHeading | pubmed-meshheading:18205950... | lld:pubmed |
| pubmed-article:18205950 | pubmed:meshHeading | pubmed-meshheading:18205950... | lld:pubmed |
| pubmed-article:18205950 | pubmed:meshHeading | pubmed-meshheading:18205950... | lld:pubmed |
| pubmed-article:18205950 | pubmed:meshHeading | pubmed-meshheading:18205950... | lld:pubmed |
| pubmed-article:18205950 | pubmed:meshHeading | pubmed-meshheading:18205950... | lld:pubmed |
| pubmed-article:18205950 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18205950 | pubmed:articleTitle | Derlin-1 is overexpressed in human breast carcinoma and protects cancer cells from endoplasmic reticulum stress-induced apoptosis. | lld:pubmed |
| pubmed-article:18205950 | pubmed:affiliation | Division of Signal Transduction and Molecular Targeting Therapy, State Key Laboratory of Biotherapy, West China Hospital, Sichuan University, No. 1 Ke Yuan 4 Lu, Chengdu, 610041, China. | lld:pubmed |
| pubmed-article:18205950 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18205950 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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