| pubmed-article:181590 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:181590 | lifeskim:mentions | umls-concept:C0042776 | lld:lifeskim |
| pubmed-article:181590 | lifeskim:mentions | umls-concept:C1623048 | lld:lifeskim |
| pubmed-article:181590 | lifeskim:mentions | umls-concept:C0013878 | lld:lifeskim |
| pubmed-article:181590 | lifeskim:mentions | umls-concept:C0596988 | lld:lifeskim |
| pubmed-article:181590 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:181590 | pubmed:dateCreated | 1976-10-2 | lld:pubmed |
| pubmed-article:181590 | pubmed:abstractText | Temperature-sensitive (ts) mutants of vesicular stomatitis virus belonging to complementation groups I, II and IV inhibited the replication of wild-type vesicular stomatitis virus when mixed infections were carried out in BHK21 cells at 32, 37, and 39.5 C. The group IV mutant (ts G 41) was most effective in this regard; wild-type virus yields were inhibited almost 1,000-fold in mixed infections with this mutant at 32 C. In the case of group I and II mutants, inhibition of wild-type virus replication at 37 and 39.5 C was accompanied by an enhancement (up to 15,000-fold) of the yields of the coinfecting ts mutant. The yields of the group IV mutant (ts G 41) were not enhanced by mixed infections with wild-type virus at any temperature, although this mutant inhibited wild-type virus replication at all temperatures. The dominance of the replication of ts mutants at 37 C provides a rationale for the selection and maintenance of ts virus in persistently infected cells. | lld:pubmed |
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| pubmed-article:181590 | pubmed:language | eng | lld:pubmed |
| pubmed-article:181590 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:181590 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:181590 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:181590 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:181590 | pubmed:issn | 0022-538X | lld:pubmed |
| pubmed-article:181590 | pubmed:author | pubmed-author:YoungnerJ SJS | lld:pubmed |
| pubmed-article:181590 | pubmed:author | pubmed-author:QuaglianaD... | lld:pubmed |
| pubmed-article:181590 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:181590 | pubmed:volume | 19 | lld:pubmed |
| pubmed-article:181590 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:181590 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:181590 | pubmed:pagination | 102-7 | lld:pubmed |
| pubmed-article:181590 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
| pubmed-article:181590 | pubmed:meshHeading | pubmed-meshheading:181590-T... | lld:pubmed |
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| pubmed-article:181590 | pubmed:meshHeading | pubmed-meshheading:181590-V... | lld:pubmed |
| pubmed-article:181590 | pubmed:meshHeading | pubmed-meshheading:181590-C... | lld:pubmed |
| pubmed-article:181590 | pubmed:meshHeading | pubmed-meshheading:181590-G... | lld:pubmed |
| pubmed-article:181590 | pubmed:meshHeading | pubmed-meshheading:181590-D... | lld:pubmed |
| pubmed-article:181590 | pubmed:year | 1976 | lld:pubmed |
| pubmed-article:181590 | pubmed:articleTitle | Temperature-sensitive mutants of vesicular stomatitis virus are conditionally defective particles that interfere with and are rescued by wild-type virus. | lld:pubmed |
| pubmed-article:181590 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:181590 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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