pubmed-article:17957240 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:17957240 | lifeskim:mentions | umls-concept:C1956346 | lld:lifeskim |
pubmed-article:17957240 | lifeskim:mentions | umls-concept:C0449438 | lld:lifeskim |
pubmed-article:17957240 | lifeskim:mentions | umls-concept:C0242599 | lld:lifeskim |
pubmed-article:17957240 | lifeskim:mentions | umls-concept:C0205360 | lld:lifeskim |
pubmed-article:17957240 | pubmed:issue | 10 | lld:pubmed |
pubmed-article:17957240 | pubmed:dateCreated | 2007-10-24 | lld:pubmed |
pubmed-article:17957240 | pubmed:abstractText | During the last years, neutrophils have emerged as important players in atherogenesis. They are highly activated in peripheral blood of patients with unstable angina. Moreover, a primed state of circulating neutrophils has been proposed in patients with stable angina. Our aim was to investigate the neutrophil activation status in patients with stable coronary artery disease (CAD) at conventional drug treatment. | lld:pubmed |
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pubmed-article:17957240 | pubmed:language | eng | lld:pubmed |
pubmed-article:17957240 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17957240 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:17957240 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:17957240 | pubmed:issn | 1932-6203 | lld:pubmed |
pubmed-article:17957240 | pubmed:author | pubmed-author:JonassonLenaL | lld:pubmed |
pubmed-article:17957240 | pubmed:author | pubmed-author:SärndahlEvaE | lld:pubmed |
pubmed-article:17957240 | pubmed:author | pubmed-author:NijmJohnnyJ | lld:pubmed |
pubmed-article:17957240 | pubmed:author | pubmed-author:BrodinVeronik... | lld:pubmed |
pubmed-article:17957240 | pubmed:author | pubmed-author:BergströmIdaI | lld:pubmed |
pubmed-article:17957240 | pubmed:author | pubmed-author:Lundqvist... | lld:pubmed |
pubmed-article:17957240 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:17957240 | pubmed:volume | 2 | lld:pubmed |
pubmed-article:17957240 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:17957240 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:17957240 | pubmed:pagination | e1056 | lld:pubmed |
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pubmed-article:17957240 | pubmed:year | 2007 | lld:pubmed |
pubmed-article:17957240 | pubmed:articleTitle | Neutrophil activation status in stable coronary artery disease. | lld:pubmed |
pubmed-article:17957240 | pubmed:affiliation | Division of Medical Microbiology, Department of Clinical and Experimental Medicine, Faculty of Health Sciences, Linköping University, Linköping, Sweden. eva.sarndahl@ikm.oru.se | lld:pubmed |
pubmed-article:17957240 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:17957240 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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