| pubmed-article:17903725 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17903725 | lifeskim:mentions | umls-concept:C0390423 | lld:lifeskim |
| pubmed-article:17903725 | lifeskim:mentions | umls-concept:C1880805 | lld:lifeskim |
| pubmed-article:17903725 | lifeskim:mentions | umls-concept:C0337108 | lld:lifeskim |
| pubmed-article:17903725 | lifeskim:mentions | umls-concept:C1999230 | lld:lifeskim |
| pubmed-article:17903725 | lifeskim:mentions | umls-concept:C0205227 | lld:lifeskim |
| pubmed-article:17903725 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:17903725 | pubmed:dateCreated | 2007-10-1 | lld:pubmed |
| pubmed-article:17903725 | pubmed:abstractText | The dual actions of angiotensin II (AngII) on microvascular fluid leak remain enigmatic. Our hypothesis was that the AngII type 2 (AT2) receptor decreases microvascular fluid leak during inflammation. The purposes of this study were to determine the activity of the AT2 receptor during stimulation by endogenous AngII, during stimulation by exogenous AngII, and during inflammation. | lld:pubmed |
| pubmed-article:17903725 | pubmed:language | eng | lld:pubmed |
| pubmed-article:17903725 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17903725 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:17903725 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17903725 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17903725 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17903725 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17903725 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17903725 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:17903725 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:17903725 | pubmed:issn | 1879-1190 | lld:pubmed |
| pubmed-article:17903725 | pubmed:author | pubmed-author:VictorinoGreg... | lld:pubmed |
| pubmed-article:17903725 | pubmed:author | pubmed-author:CurranBrianB | lld:pubmed |
| pubmed-article:17903725 | pubmed:author | pubmed-author:SadjadiJavidJ | lld:pubmed |
| pubmed-article:17903725 | pubmed:author | pubmed-author:EresoAlexande... | lld:pubmed |
| pubmed-article:17903725 | pubmed:author | pubmed-author:RamirezRené... | lld:pubmed |
| pubmed-article:17903725 | pubmed:author | pubmed-author:CrippsMichael... | lld:pubmed |
| pubmed-article:17903725 | pubmed:author | pubmed-author:CuretonElizab... | lld:pubmed |
| pubmed-article:17903725 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:17903725 | pubmed:volume | 205 | lld:pubmed |
| pubmed-article:17903725 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:17903725 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:17903725 | pubmed:pagination | 527-33 | lld:pubmed |
| pubmed-article:17903725 | pubmed:dateRevised | 2009-5-21 | lld:pubmed |
| pubmed-article:17903725 | pubmed:meshHeading | pubmed-meshheading:17903725... | lld:pubmed |
| pubmed-article:17903725 | pubmed:meshHeading | pubmed-meshheading:17903725... | lld:pubmed |
| pubmed-article:17903725 | pubmed:meshHeading | pubmed-meshheading:17903725... | lld:pubmed |
| pubmed-article:17903725 | pubmed:meshHeading | pubmed-meshheading:17903725... | lld:pubmed |
| pubmed-article:17903725 | pubmed:meshHeading | pubmed-meshheading:17903725... | lld:pubmed |
| pubmed-article:17903725 | pubmed:meshHeading | pubmed-meshheading:17903725... | lld:pubmed |
| pubmed-article:17903725 | pubmed:meshHeading | pubmed-meshheading:17903725... | lld:pubmed |
| pubmed-article:17903725 | pubmed:meshHeading | pubmed-meshheading:17903725... | lld:pubmed |
| pubmed-article:17903725 | pubmed:meshHeading | pubmed-meshheading:17903725... | lld:pubmed |
| pubmed-article:17903725 | pubmed:meshHeading | pubmed-meshheading:17903725... | lld:pubmed |
| pubmed-article:17903725 | pubmed:meshHeading | pubmed-meshheading:17903725... | lld:pubmed |
| pubmed-article:17903725 | pubmed:meshHeading | pubmed-meshheading:17903725... | lld:pubmed |
| pubmed-article:17903725 | pubmed:meshHeading | pubmed-meshheading:17903725... | lld:pubmed |
| pubmed-article:17903725 | pubmed:meshHeading | pubmed-meshheading:17903725... | lld:pubmed |
| pubmed-article:17903725 | pubmed:meshHeading | pubmed-meshheading:17903725... | lld:pubmed |
| pubmed-article:17903725 | pubmed:year | 2007 | lld:pubmed |
| pubmed-article:17903725 | pubmed:articleTitle | Angiotensin II type 2 receptor provides an endogenous brake during inflammation-induced microvascular fluid leak. | lld:pubmed |
| pubmed-article:17903725 | pubmed:affiliation | Department of Surgery, UCSF-East Bay, Alameda County Medical Center, Oakland, CA 94602, USA. | lld:pubmed |
| pubmed-article:17903725 | pubmed:publicationType | Journal Article | lld:pubmed |
