| pubmed-article:17707074 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17707074 | lifeskim:mentions | umls-concept:C0028128 | lld:lifeskim |
| pubmed-article:17707074 | lifeskim:mentions | umls-concept:C0032105 | lld:lifeskim |
| pubmed-article:17707074 | lifeskim:mentions | umls-concept:C0007578 | lld:lifeskim |
| pubmed-article:17707074 | lifeskim:mentions | umls-concept:C0441889 | lld:lifeskim |
| pubmed-article:17707074 | lifeskim:mentions | umls-concept:C0919427 | lld:lifeskim |
| pubmed-article:17707074 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
| pubmed-article:17707074 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:17707074 | pubmed:dateCreated | 2008-2-5 | lld:pubmed |
| pubmed-article:17707074 | pubmed:abstractText | Hyperhomocysteinemia is an independent risk factor for cardiovascular events. The T allele of 677 C/T polymorphism at the methylenetetrahydrofolate reductase (MTHFR) gene has been reported to induce mild hyperhomocysteinemia. In the present study, we investigated the relationship between this polymorphism and adhesion molecules and total nitric oxide (NOx). | lld:pubmed |
| pubmed-article:17707074 | pubmed:language | eng | lld:pubmed |
| pubmed-article:17707074 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17707074 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:17707074 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:17707074 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17707074 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:17707074 | pubmed:issn | 0049-3848 | lld:pubmed |
| pubmed-article:17707074 | pubmed:author | pubmed-author:JuoSuh-Hang... | lld:pubmed |
| pubmed-article:17707074 | pubmed:author | pubmed-author:LiuChin-SanCS | lld:pubmed |
| pubmed-article:17707074 | pubmed:author | pubmed-author:HuangChing-Sh... | lld:pubmed |
| pubmed-article:17707074 | pubmed:author | pubmed-author:KuoChen-LingC... | lld:pubmed |
| pubmed-article:17707074 | pubmed:author | pubmed-author:LiaoYi-ChuYC | lld:pubmed |
| pubmed-article:17707074 | pubmed:author | pubmed-author:ChiangHui-Chi... | lld:pubmed |
| pubmed-article:17707074 | pubmed:author | pubmed-author:WangYihsinY | lld:pubmed |
| pubmed-article:17707074 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:17707074 | pubmed:volume | 121 | lld:pubmed |
| pubmed-article:17707074 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:17707074 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:17707074 | pubmed:pagination | 549-54 | lld:pubmed |
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| pubmed-article:17707074 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:17707074 | pubmed:articleTitle | The MTHFR 677 C/T polymorphism influences plasma levels of adhesion molecules and nitric oxide. | lld:pubmed |
| pubmed-article:17707074 | pubmed:affiliation | Graduate Institute of Medical Genetics, Kaohsiung Medical University, Department of Medical Research, Kaohsiung Medical University Hospital, Kaohsiung, Taiwan. | lld:pubmed |
| pubmed-article:17707074 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:17707074 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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| http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:17707074 | lld:entrezgene |
