pubmed-article:17618 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:17618 | lifeskim:mentions | umls-concept:C0014819 | lld:lifeskim |
pubmed-article:17618 | lifeskim:mentions | umls-concept:C0001644 | lld:lifeskim |
pubmed-article:17618 | lifeskim:mentions | umls-concept:C1947989 | lld:lifeskim |
pubmed-article:17618 | lifeskim:mentions | umls-concept:C1709059 | lld:lifeskim |
pubmed-article:17618 | lifeskim:mentions | umls-concept:C1533691 | lld:lifeskim |
pubmed-article:17618 | lifeskim:mentions | umls-concept:C1522492 | lld:lifeskim |
pubmed-article:17618 | lifeskim:mentions | umls-concept:C1516960 | lld:lifeskim |
pubmed-article:17618 | pubmed:issue | 1 | lld:pubmed |
pubmed-article:17618 | pubmed:dateCreated | 1977-8-12 | lld:pubmed |
pubmed-article:17618 | pubmed:abstractText | Canine marrow erythroid colony growth is enhanced by agents linked to the adenyl cyclase/cyclic AMP (cAMP) system, including cAMP, a phosphodieterase inhibitor (RO-20-1724), cholera enterotoxin, and beta-adrenergic agonists. The adrenergic effect is mediated by receptors having beta2-subspecificity. These receptors are distinct from putative receptors for erythropoietin and those acted upon by cholera enterotoxin. In addition, the population of cells most responsive to beta-agonists is distinct from the majority of erythropoientin-responsive cells, perhaps representing a subpopulation of this class of cell. This demonstration of an adenyl cyclase-linked mechanism regulating mammalian erythroid colony growth provides a model for the modulation by other hormones or small molecules of in vitro and, perhaps, in vivo erythropoiesis. | lld:pubmed |
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pubmed-article:17618 | pubmed:language | eng | lld:pubmed |
pubmed-article:17618 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17618 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:17618 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:17618 | pubmed:month | Jul | lld:pubmed |
pubmed-article:17618 | pubmed:issn | 0021-9738 | lld:pubmed |
pubmed-article:17618 | pubmed:author | pubmed-author:AdamsonJ WJW | lld:pubmed |
pubmed-article:17618 | pubmed:author | pubmed-author:BrownJ EJE | lld:pubmed |
pubmed-article:17618 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:17618 | pubmed:volume | 60 | lld:pubmed |
pubmed-article:17618 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:17618 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:17618 | pubmed:pagination | 70-7 | lld:pubmed |
pubmed-article:17618 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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pubmed-article:17618 | pubmed:year | 1977 | lld:pubmed |
pubmed-article:17618 | pubmed:articleTitle | Modulation of in vitro erythropoiesis. The influence of beta-adrenergic agonists on erythroid colony formation. | lld:pubmed |
pubmed-article:17618 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:17618 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:17618 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
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