| pubmed-article:17618301 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17618301 | lifeskim:mentions | umls-concept:C0012356 | lld:lifeskim |
| pubmed-article:17618301 | lifeskim:mentions | umls-concept:C0205042 | lld:lifeskim |
| pubmed-article:17618301 | lifeskim:mentions | umls-concept:C0086045 | lld:lifeskim |
| pubmed-article:17618301 | lifeskim:mentions | umls-concept:C0012359 | lld:lifeskim |
| pubmed-article:17618301 | lifeskim:mentions | umls-concept:C0244404 | lld:lifeskim |
| pubmed-article:17618301 | lifeskim:mentions | umls-concept:C0302350 | lld:lifeskim |
| pubmed-article:17618301 | lifeskim:mentions | umls-concept:C1533691 | lld:lifeskim |
| pubmed-article:17618301 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:17618301 | pubmed:dateCreated | 2007-9-10 | lld:pubmed |
| pubmed-article:17618301 | pubmed:abstractText | Raloxifene improves cardiovascular function. This study examines the hypothesis that therapeutic concentrations of raloxifene augment endothelium-dependent relaxation via up-regulation of eNOS expression and activity in porcine coronary arteries. | lld:pubmed |
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| pubmed-article:17618301 | pubmed:language | eng | lld:pubmed |
| pubmed-article:17618301 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17618301 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:17618301 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:17618301 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:17618301 | pubmed:issn | 0007-1188 | lld:pubmed |
| pubmed-article:17618301 | pubmed:author | pubmed-author:VanhoutteP... | lld:pubmed |
| pubmed-article:17618301 | pubmed:author | pubmed-author:HuangYY | lld:pubmed |
| pubmed-article:17618301 | pubmed:author | pubmed-author:AuC LCL | lld:pubmed |
| pubmed-article:17618301 | pubmed:author | pubmed-author:YanYY | lld:pubmed |
| pubmed-article:17618301 | pubmed:author | pubmed-author:LaherII | lld:pubmed |
| pubmed-article:17618301 | pubmed:author | pubmed-author:LeungH SHS | lld:pubmed |
| pubmed-article:17618301 | pubmed:author | pubmed-author:YungL MLM | lld:pubmed |
| pubmed-article:17618301 | pubmed:author | pubmed-author:LeungF PFP | lld:pubmed |
| pubmed-article:17618301 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:17618301 | pubmed:volume | 152 | lld:pubmed |
| pubmed-article:17618301 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:17618301 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:17618301 | pubmed:pagination | 223-9 | lld:pubmed |
| pubmed-article:17618301 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:17618301 | pubmed:meshHeading | pubmed-meshheading:17618301... | lld:pubmed |
| pubmed-article:17618301 | pubmed:meshHeading | pubmed-meshheading:17618301... | lld:pubmed |
| pubmed-article:17618301 | pubmed:meshHeading | pubmed-meshheading:17618301... | lld:pubmed |
| pubmed-article:17618301 | pubmed:meshHeading | pubmed-meshheading:17618301... | lld:pubmed |
| pubmed-article:17618301 | pubmed:meshHeading | pubmed-meshheading:17618301... | lld:pubmed |
| pubmed-article:17618301 | pubmed:year | 2007 | lld:pubmed |
| pubmed-article:17618301 | pubmed:articleTitle | Therapeutic concentrations of raloxifene augment nitric oxide-dependent coronary artery dilatation in vitro. | lld:pubmed |
| pubmed-article:17618301 | pubmed:affiliation | Li Ka Shing Institute of Health Sciences, Chinese University of Hong Kong, Hong Kong SAR, China. | lld:pubmed |
| pubmed-article:17618301 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:17618301 | pubmed:publicationType | In Vitro | lld:pubmed |
| pubmed-article:17618301 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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