pubmed-article:17615386 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:17615386 | lifeskim:mentions | umls-concept:C0214743 | lld:lifeskim |
pubmed-article:17615386 | lifeskim:mentions | umls-concept:C0035245 | lld:lifeskim |
pubmed-article:17615386 | lifeskim:mentions | umls-concept:C0559546 | lld:lifeskim |
pubmed-article:17615386 | lifeskim:mentions | umls-concept:C0453996 | lld:lifeskim |
pubmed-article:17615386 | lifeskim:mentions | umls-concept:C0086860 | lld:lifeskim |
pubmed-article:17615386 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
pubmed-article:17615386 | pubmed:issue | 8 | lld:pubmed |
pubmed-article:17615386 | pubmed:dateCreated | 2007-10-4 | lld:pubmed |
pubmed-article:17615386 | pubmed:abstractText | Although the duration and amount of cigarette smoking correlate with reduction in pulmonary function, there is still variation among individual responses. IL-13 is involved in pulmonary inflammation, remodeling, and susceptibility to chronic obstructive pulmonary disease (COPD). | lld:pubmed |
pubmed-article:17615386 | pubmed:language | eng | lld:pubmed |
pubmed-article:17615386 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17615386 | pubmed:citationSubset | AIM | lld:pubmed |
pubmed-article:17615386 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17615386 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:17615386 | pubmed:month | Oct | lld:pubmed |
pubmed-article:17615386 | pubmed:issn | 1073-449X | lld:pubmed |
pubmed-article:17615386 | pubmed:author | pubmed-author:MeyersDeborah... | lld:pubmed |
pubmed-article:17615386 | pubmed:author | pubmed-author:BleeckerEugen... | lld:pubmed |
pubmed-article:17615386 | pubmed:author | pubmed-author:SterlingDavid... | lld:pubmed |
pubmed-article:17615386 | pubmed:author | pubmed-author:BottaiMatteoM | lld:pubmed |
pubmed-article:17615386 | pubmed:author | pubmed-author:OharJill AJA | lld:pubmed |
pubmed-article:17615386 | pubmed:author | pubmed-author:SadeghnejadAl... | lld:pubmed |
pubmed-article:17615386 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:17615386 | pubmed:day | 15 | lld:pubmed |
pubmed-article:17615386 | pubmed:volume | 176 | lld:pubmed |
pubmed-article:17615386 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:17615386 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:17615386 | pubmed:pagination | 748-52 | lld:pubmed |
pubmed-article:17615386 | pubmed:dateRevised | 2009-11-3 | lld:pubmed |
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pubmed-article:17615386 | pubmed:meshHeading | pubmed-meshheading:17615386... | lld:pubmed |
pubmed-article:17615386 | pubmed:year | 2007 | lld:pubmed |
pubmed-article:17615386 | pubmed:articleTitle | IL13 promoter polymorphism 1112C/T modulates the adverse effect of tobacco smoking on lung function. | lld:pubmed |
pubmed-article:17615386 | pubmed:affiliation | Center for Human Genomics and Department of Medicine, Wake Forest University School of Medicine, Medical Center Blvd., Winston-Salem, NC 27157, USA. asadeghn@wfubmc.edu | lld:pubmed |
pubmed-article:17615386 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:17615386 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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