pubmed-article:17511889 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:17511889 | lifeskim:mentions | umls-concept:C0043210 | lld:lifeskim |
pubmed-article:17511889 | lifeskim:mentions | umls-concept:C0041427 | lld:lifeskim |
pubmed-article:17511889 | lifeskim:mentions | umls-concept:C0337514 | lld:lifeskim |
pubmed-article:17511889 | lifeskim:mentions | umls-concept:C0170657 | lld:lifeskim |
pubmed-article:17511889 | lifeskim:mentions | umls-concept:C1167622 | lld:lifeskim |
pubmed-article:17511889 | lifeskim:mentions | umls-concept:C2603343 | lld:lifeskim |
pubmed-article:17511889 | pubmed:dateCreated | 2007-6-13 | lld:pubmed |
pubmed-article:17511889 | pubmed:abstractText | Bulimia Nervosa (BN) is believed to be caused by an interaction of genetic and environmental factors. Previous studies support the existence of a bulimia-related endophenotype as well as disturbances in serotonin (5-HT) transmission. We studied serotonin transporter (SERT) binding in BN, and to investigate the possibility of a SERT-related endophenotype for BN, did this in a sample of female twins. We hypothesized clearly reduced SERT binding in BN women as opposed to healthy women, and intermediate SERT binding in unaffected co-twins. | lld:pubmed |
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pubmed-article:17511889 | pubmed:language | eng | lld:pubmed |
pubmed-article:17511889 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17511889 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:17511889 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:17511889 | pubmed:issn | 1471-244X | lld:pubmed |
pubmed-article:17511889 | pubmed:author | pubmed-author:RissanenAilaA | lld:pubmed |
pubmed-article:17511889 | pubmed:author | pubmed-author:KaprioJaakkoJ | lld:pubmed |
pubmed-article:17511889 | pubmed:author | pubmed-author:AhonenAapoA | lld:pubmed |
pubmed-article:17511889 | pubmed:author | pubmed-author:KauppinenTomi... | lld:pubmed |
pubmed-article:17511889 | pubmed:author | pubmed-author:Keski-Rahkone... | lld:pubmed |
pubmed-article:17511889 | pubmed:author | pubmed-author:SihvolaElinaE | lld:pubmed |
pubmed-article:17511889 | pubmed:author | pubmed-author:KoskelaAnu... | lld:pubmed |
pubmed-article:17511889 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:17511889 | pubmed:volume | 7 | lld:pubmed |
pubmed-article:17511889 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:17511889 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:17511889 | pubmed:pagination | 19 | lld:pubmed |
pubmed-article:17511889 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
pubmed-article:17511889 | pubmed:meshHeading | pubmed-meshheading:17511889... | lld:pubmed |
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pubmed-article:17511889 | pubmed:meshHeading | pubmed-meshheading:17511889... | lld:pubmed |
pubmed-article:17511889 | pubmed:year | 2007 | lld:pubmed |
pubmed-article:17511889 | pubmed:articleTitle | Serotonin transporter binding of [123I]ADAM in bulimic women, their healthy twin sisters, and healthy women: a SPET study. | lld:pubmed |
pubmed-article:17511889 | pubmed:affiliation | Department of Clinical Physiology and Nuclear Medicine, HUSLAB/Helsinki University Central Hospital, Helsinki, Finland. anu.koskela@helsinki.fi | lld:pubmed |
pubmed-article:17511889 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:17511889 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |