| pubmed-article:17505012 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17505012 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
| pubmed-article:17505012 | lifeskim:mentions | umls-concept:C0040690 | lld:lifeskim |
| pubmed-article:17505012 | lifeskim:mentions | umls-concept:C0021467 | lld:lifeskim |
| pubmed-article:17505012 | lifeskim:mentions | umls-concept:C0021469 | lld:lifeskim |
| pubmed-article:17505012 | pubmed:issue | 10 | lld:pubmed |
| pubmed-article:17505012 | pubmed:dateCreated | 2007-5-16 | lld:pubmed |
| pubmed-article:17505012 | pubmed:abstractText | Transforming growth factor beta (TGF-beta), which generally stimulates the growth of mesenchymally derived cells but inhibits the growth of epithelial cells, has been proposed as a possible target for cancer therapy. However, concerns have been raised that whereas inhibition of TGF-beta signaling could be efficacious for lesions in which TGF-beta promotes tumor development and/or progression, systemic pharmacologic blockade of this signaling pathway could also promote the growth of epithelial lesions. | lld:pubmed |
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| pubmed-article:17505012 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:17505012 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:17505012 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17505012 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:17505012 | pubmed:month | May | lld:pubmed |
| pubmed-article:17505012 | pubmed:issn | 1078-0432 | lld:pubmed |
| pubmed-article:17505012 | pubmed:author | pubmed-author:LapingNichola... | lld:pubmed |
| pubmed-article:17505012 | pubmed:author | pubmed-author:EverittJeffre... | lld:pubmed |
| pubmed-article:17505012 | pubmed:author | pubmed-author:WalkerCheryl... | lld:pubmed |
| pubmed-article:17505012 | pubmed:author | pubmed-author:GoldLeslie... | lld:pubmed |
| pubmed-article:17505012 | pubmed:author | pubmed-author:FrazierKendal... | lld:pubmed |
| pubmed-article:17505012 | pubmed:author | pubmed-author:BurgertMarkM | lld:pubmed |
| pubmed-article:17505012 | pubmed:author | pubmed-author:PortisMelisa... | lld:pubmed |
| pubmed-article:17505012 | pubmed:author | pubmed-author:CadacioCapric... | lld:pubmed |
| pubmed-article:17505012 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:17505012 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:17505012 | pubmed:volume | 13 | lld:pubmed |
| pubmed-article:17505012 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:17505012 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:17505012 | pubmed:pagination | 3087-99 | lld:pubmed |
| pubmed-article:17505012 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:17505012 | pubmed:meshHeading | pubmed-meshheading:17505012... | lld:pubmed |
| pubmed-article:17505012 | pubmed:year | 2007 | lld:pubmed |
| pubmed-article:17505012 | pubmed:articleTitle | Tumor-specific efficacy of transforming growth factor-beta RI inhibition in Eker rats. | lld:pubmed |
| pubmed-article:17505012 | pubmed:affiliation | GlaxoSmithKline Pharmaceuticals, King of Prussia, Pennsylvania, USA. | lld:pubmed |
| pubmed-article:17505012 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:17505012 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:17505012 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
| entrez-gene:29591 | entrezgene:pubmed | pubmed-article:17505012 | lld:entrezgene |
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