pubmed-article:17487826 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:17487826 | lifeskim:mentions | umls-concept:C0379886 | lld:lifeskim |
pubmed-article:17487826 | lifeskim:mentions | umls-concept:C1704898 | lld:lifeskim |
pubmed-article:17487826 | lifeskim:mentions | umls-concept:C0003018 | lld:lifeskim |
pubmed-article:17487826 | lifeskim:mentions | umls-concept:C0024432 | lld:lifeskim |
pubmed-article:17487826 | lifeskim:mentions | umls-concept:C1332693 | lld:lifeskim |
pubmed-article:17487826 | lifeskim:mentions | umls-concept:C1522249 | lld:lifeskim |
pubmed-article:17487826 | lifeskim:mentions | umls-concept:C0392747 | lld:lifeskim |
pubmed-article:17487826 | lifeskim:mentions | umls-concept:C1948023 | lld:lifeskim |
pubmed-article:17487826 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
pubmed-article:17487826 | lifeskim:mentions | umls-concept:C1956267 | lld:lifeskim |
pubmed-article:17487826 | pubmed:issue | 1 | lld:pubmed |
pubmed-article:17487826 | pubmed:dateCreated | 2007-5-9 | lld:pubmed |
pubmed-article:17487826 | pubmed:abstractText | Macrophages play critical roles in the development of atherosclerosis and diabetic nephropathy as well as many inflammatory diseases. Angiotensin II type 1 receptor antagonists (AIIA) are beneficial for the prevention of atherosclerosis and diabetic nephropathy suggesting that angiotensin II (Ang II) promotes the development of these diseases. It has recently been reported that Ang II exerts proinflammatory actions in vivo and in vitro. This study was aimed to clarify the direct effects of Ang II on monocytes/macrophages. | lld:pubmed |
pubmed-article:17487826 | pubmed:language | eng | lld:pubmed |
pubmed-article:17487826 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17487826 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:17487826 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17487826 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:17487826 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17487826 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:17487826 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17487826 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:17487826 | pubmed:month | Mar | lld:pubmed |
pubmed-article:17487826 | pubmed:issn | 1470-3203 | lld:pubmed |
pubmed-article:17487826 | pubmed:author | pubmed-author:WildD JDJ | lld:pubmed |
pubmed-article:17487826 | pubmed:author | pubmed-author:MakinoHirofum... | lld:pubmed |
pubmed-article:17487826 | pubmed:author | pubmed-author:ShikataKenich... | lld:pubmed |
pubmed-article:17487826 | pubmed:author | pubmed-author:OkadaShinichi... | lld:pubmed |
pubmed-article:17487826 | pubmed:author | pubmed-author:OgawaDaisukeD | lld:pubmed |
pubmed-article:17487826 | pubmed:author | pubmed-author:ShikataYasush... | lld:pubmed |
pubmed-article:17487826 | pubmed:author | pubmed-author:ToneAtsuhitoA | lld:pubmed |
pubmed-article:17487826 | pubmed:author | pubmed-author:SasakiSakikoS | lld:pubmed |
pubmed-article:17487826 | pubmed:author | pubmed-author:NagaseRyoR | lld:pubmed |
pubmed-article:17487826 | pubmed:author | pubmed-author:SasakiMotofum... | lld:pubmed |
pubmed-article:17487826 | pubmed:author | pubmed-author:YozaiKosukeK | lld:pubmed |
pubmed-article:17487826 | pubmed:author | pubmed-author:UsuiHitomi... | lld:pubmed |
pubmed-article:17487826 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:17487826 | pubmed:volume | 8 | lld:pubmed |
pubmed-article:17487826 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:17487826 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:17487826 | pubmed:pagination | 45-50 | lld:pubmed |
pubmed-article:17487826 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
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pubmed-article:17487826 | pubmed:year | 2007 | lld:pubmed |
pubmed-article:17487826 | pubmed:articleTitle | Changes of gene expression profiles in macrophages stimulated by angiotensin II--angiotensin II induces MCP-2 through AT1-receptor. | lld:pubmed |
pubmed-article:17487826 | pubmed:affiliation | Department of Medicine and Clinical Science, Okayama University Graduate School of Medicine, Dentistry, and Pharmaceutical Sciences, Okayama, Japan. | lld:pubmed |
pubmed-article:17487826 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:17487826 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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