| pubmed-article:17479278 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17479278 | lifeskim:mentions | umls-concept:C1527116 | lld:lifeskim |
| pubmed-article:17479278 | lifeskim:mentions | umls-concept:C0035647 | lld:lifeskim |
| pubmed-article:17479278 | lifeskim:mentions | umls-concept:C0014442 | lld:lifeskim |
| pubmed-article:17479278 | lifeskim:mentions | umls-concept:C0007134 | lld:lifeskim |
| pubmed-article:17479278 | lifeskim:mentions | umls-concept:C0311400 | lld:lifeskim |
| pubmed-article:17479278 | lifeskim:mentions | umls-concept:C0596244 | lld:lifeskim |
| pubmed-article:17479278 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
| pubmed-article:17479278 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:17479278 | pubmed:dateCreated | 2007-10-17 | lld:pubmed |
| pubmed-article:17479278 | pubmed:abstractText | Individual differences in susceptibility to trichloroethylene-induced nephrocarcinogenicity may be conferred by genetic polymorphisms of glutathione S-transferases (GST), because enzymes of this group are pivotal for the metabolic activation of trichloroethylene. Because of a potential involvement of N-acetylation in the detoxication of reactive trichloroethylene metabolite(s) to N-acetyl-cysteine derivatives, polymorphisms of the NAT2 gene may also be relevant. | lld:pubmed |
| pubmed-article:17479278 | pubmed:language | eng | lld:pubmed |
| pubmed-article:17479278 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17479278 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:17479278 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:17479278 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:17479278 | pubmed:issn | 0340-0131 | lld:pubmed |
| pubmed-article:17479278 | pubmed:author | pubmed-author:BoltHermann... | lld:pubmed |
| pubmed-article:17479278 | pubmed:author | pubmed-author:GolkaKlausK | lld:pubmed |
| pubmed-article:17479278 | pubmed:author | pubmed-author:BrüningThomas... | lld:pubmed |
| pubmed-article:17479278 | pubmed:author | pubmed-author:SelinskiSilvi... | lld:pubmed |
| pubmed-article:17479278 | pubmed:author | pubmed-author:WiesenhütterB... | lld:pubmed |
| pubmed-article:17479278 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:17479278 | pubmed:volume | 81 | lld:pubmed |
| pubmed-article:17479278 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:17479278 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:17479278 | pubmed:pagination | 247-51 | lld:pubmed |
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| pubmed-article:17479278 | pubmed:meshHeading | pubmed-meshheading:17479278... | lld:pubmed |
| pubmed-article:17479278 | pubmed:year | 2007 | lld:pubmed |
| pubmed-article:17479278 | pubmed:articleTitle | Re-assessment of the influence of polymorphisms of phase-II metabolic enzymes on renal cell cancer risk of trichloroethylene-exposed workers. | lld:pubmed |
| pubmed-article:17479278 | pubmed:affiliation | Institut für Arbeitsphysiologie an der Universität Dortmund, Ardeystr. 67, 44139 Dortmund, Germany. | lld:pubmed |
| pubmed-article:17479278 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:17479278 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:17479278 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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