| pubmed-article:17446858 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17446858 | lifeskim:mentions | umls-concept:C0006142 | lld:lifeskim |
| pubmed-article:17446858 | lifeskim:mentions | umls-concept:C0026339 | lld:lifeskim |
| pubmed-article:17446858 | lifeskim:mentions | umls-concept:C0025936 | lld:lifeskim |
| pubmed-article:17446858 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
| pubmed-article:17446858 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:17446858 | pubmed:dateCreated | 2007-4-25 | lld:pubmed |
| pubmed-article:17446858 | pubmed:abstractText | One-third of patients with breast cancer overexpress the ERBB2 receptor tyrosine kinase, which is associated not only with a more aggressive phenotype but also reduced responsiveness to hormonal therapies. Over the past two decades, many ERBB2 mouse models for breast cancer have conclusively shown that this receptor has a causal role in breast cancer development. These mouse models have also enabled the mechanisms controlling tumour growth, angiogenesis, metastasis, dormancy and recurrence in ERBB2-positive breast cancer to be elucidated. In addition, a mouse model has recently been described that accurately recapitulates many of the hallmarks associated with the early stages of the human disease. | lld:pubmed |
| pubmed-article:17446858 | pubmed:language | eng | lld:pubmed |
| pubmed-article:17446858 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17446858 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:17446858 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17446858 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:17446858 | pubmed:month | May | lld:pubmed |
| pubmed-article:17446858 | pubmed:issn | 1474-175X | lld:pubmed |
| pubmed-article:17446858 | pubmed:author | pubmed-author:CardiffRobert... | lld:pubmed |
| pubmed-article:17446858 | pubmed:author | pubmed-author:MullerWilliam... | lld:pubmed |
| pubmed-article:17446858 | pubmed:author | pubmed-author:Ursini-Siegel... | lld:pubmed |
| pubmed-article:17446858 | pubmed:author | pubmed-author:SchadeBabette... | lld:pubmed |
| pubmed-article:17446858 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:17446858 | pubmed:volume | 7 | lld:pubmed |
| pubmed-article:17446858 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:17446858 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:17446858 | pubmed:pagination | 389-97 | lld:pubmed |
| pubmed-article:17446858 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:17446858 | pubmed:year | 2007 | lld:pubmed |
| pubmed-article:17446858 | pubmed:articleTitle | Insights from transgenic mouse models of ERBB2-induced breast cancer. | lld:pubmed |
| pubmed-article:17446858 | pubmed:affiliation | Departments of Medicine and Biochemistry, McGill University, Montreal, Quebec, Canada. | lld:pubmed |
| pubmed-article:17446858 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:17446858 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:17446858 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
| pubmed-article:17446858 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:17446858 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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