pubmed-article:17392581 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:17392581 | lifeskim:mentions | umls-concept:C0085295 | lld:lifeskim |
pubmed-article:17392581 | lifeskim:mentions | umls-concept:C0021390 | lld:lifeskim |
pubmed-article:17392581 | lifeskim:mentions | umls-concept:C0683149 | lld:lifeskim |
pubmed-article:17392581 | lifeskim:mentions | umls-concept:C0277785 | lld:lifeskim |
pubmed-article:17392581 | lifeskim:mentions | umls-concept:C2004454 | lld:lifeskim |
pubmed-article:17392581 | lifeskim:mentions | umls-concept:C0205217 | lld:lifeskim |
pubmed-article:17392581 | lifeskim:mentions | umls-concept:C0205390 | lld:lifeskim |
pubmed-article:17392581 | pubmed:issue | 6 | lld:pubmed |
pubmed-article:17392581 | pubmed:dateCreated | 2007-3-29 | lld:pubmed |
pubmed-article:17392581 | pubmed:abstractText | Using a specific enzyme-linked immunosorbent assay, IL-10 concentrations were measured in serum from 62 patients with ulcerative colitis (UC), 43 with Crohn's disease (CD), 25 with other colitides, and 44 normal control subjects. Serum IL-10 concentrations were increased in patients with active UC but not in those with active CD when compared with normal control subjects. A time course study showed that in patients with UC and CD, serum concentrations of IL-6 and C-reactive protein increased during the acute phase and returned to normal as patients go into remission. Notably, serum IL-10 concentrations increased during the phase of disease resolution and declined thereafter regardless of the treatment modality. Gel filtration analysis indicated that IL-10 circulated predominantly as a dimer. In conclusion, this study shows that serum IL-10 is increased during disease recovery in patients with inflammatory bowel disease, and may be a helpful marker in monitoring disease status. | lld:pubmed |
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pubmed-article:17392581 | pubmed:language | eng | lld:pubmed |
pubmed-article:17392581 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17392581 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:17392581 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:17392581 | pubmed:issn | 0962-9351 | lld:pubmed |
pubmed-article:17392581 | pubmed:author | pubmed-author:MitsuyamaKeii... | lld:pubmed |
pubmed-article:17392581 | pubmed:author | pubmed-author:SataMichioM | lld:pubmed |
pubmed-article:17392581 | pubmed:author | pubmed-author:YamasakiHiros... | lld:pubmed |
pubmed-article:17392581 | pubmed:author | pubmed-author:TomiyasuNobuo... | lld:pubmed |
pubmed-article:17392581 | pubmed:author | pubmed-author:TsurutaOsamuO | lld:pubmed |
pubmed-article:17392581 | pubmed:author | pubmed-author:TakakiKosukeK | lld:pubmed |
pubmed-article:17392581 | pubmed:author | pubmed-author:KuwakiKotaroK | lld:pubmed |
pubmed-article:17392581 | pubmed:author | pubmed-author:MasudaJunyaJ | lld:pubmed |
pubmed-article:17392581 | pubmed:author | pubmed-author:KitazakiShige... | lld:pubmed |
pubmed-article:17392581 | pubmed:author | pubmed-author:TakedaTeikoT | lld:pubmed |
pubmed-article:17392581 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:17392581 | pubmed:volume | 2006 | lld:pubmed |
pubmed-article:17392581 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:17392581 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:17392581 | pubmed:pagination | 26875 | lld:pubmed |
pubmed-article:17392581 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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pubmed-article:17392581 | pubmed:year | 2006 | lld:pubmed |
pubmed-article:17392581 | pubmed:articleTitle | Interleukin-10 in the pathophysiology of inflammatory bowel disease: increased serum concentrations during the recovery phase. | lld:pubmed |
pubmed-article:17392581 | pubmed:affiliation | Division of Gastroenterology, Department of Medicine, Kurume University School of Medicine, Japan. | lld:pubmed |
pubmed-article:17392581 | pubmed:publicationType | Journal Article | lld:pubmed |
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