| pubmed-article:17383196 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17383196 | lifeskim:mentions | umls-concept:C1155046 | lld:lifeskim |
| pubmed-article:17383196 | lifeskim:mentions | umls-concept:C1704410 | lld:lifeskim |
| pubmed-article:17383196 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:17383196 | pubmed:dateCreated | 2007-6-1 | lld:pubmed |
| pubmed-article:17383196 | pubmed:abstractText | Several events control the activation, proliferation, and the continued Ag responsiveness of naïve and memory T lymphocytes. Here we review the individual contributions of TCR, CD28, and IL-2-driven signaling to T cell proliferation and anergy avoidance. The role of mTOR as a rheostat capable of integrating extracellular, plasma membrane-associated, and intracellular signals with relevance to T cell priming and tolerance is discussed. | lld:pubmed |
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