| pubmed-article:17343740 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17343740 | lifeskim:mentions | umls-concept:C0376358 | lld:lifeskim |
| pubmed-article:17343740 | lifeskim:mentions | umls-concept:C0069676 | lld:lifeskim |
| pubmed-article:17343740 | lifeskim:mentions | umls-concept:C0334227 | lld:lifeskim |
| pubmed-article:17343740 | lifeskim:mentions | umls-concept:C1332712 | lld:lifeskim |
| pubmed-article:17343740 | lifeskim:mentions | umls-concept:C1522484 | lld:lifeskim |
| pubmed-article:17343740 | lifeskim:mentions | umls-concept:C0036525 | lld:lifeskim |
| pubmed-article:17343740 | lifeskim:mentions | umls-concept:C0441712 | lld:lifeskim |
| pubmed-article:17343740 | pubmed:dateCreated | 2007-3-16 | lld:pubmed |
| pubmed-article:17343740 | pubmed:abstractText | The expression level of osteopontin correlates with the metastatic potential of several tumors. Osteopontin is a well-characterized ligand for the alphavbeta3 integrin. The present study was undertaken to elucidate the possible role of osteopontin/alphavbeta3 signaling in prostate cancer cell migration. | lld:pubmed |
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| pubmed-article:17343740 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:17343740 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:17343740 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:17343740 | pubmed:issn | 1476-4598 | lld:pubmed |
| pubmed-article:17343740 | pubmed:author | pubmed-author:RogersMichael... | lld:pubmed |
| pubmed-article:17343740 | pubmed:author | pubmed-author:DesaiBhavikB | lld:pubmed |
| pubmed-article:17343740 | pubmed:author | pubmed-author:ChellaiahMeen... | lld:pubmed |
| pubmed-article:17343740 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:17343740 | pubmed:volume | 6 | lld:pubmed |
| pubmed-article:17343740 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:17343740 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:17343740 | pubmed:pagination | 18 | lld:pubmed |
