| pubmed-article:17310371 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17310371 | lifeskim:mentions | umls-concept:C0011854 | lld:lifeskim |
| pubmed-article:17310371 | lifeskim:mentions | umls-concept:C1858460 | lld:lifeskim |
| pubmed-article:17310371 | lifeskim:mentions | umls-concept:C0019721 | lld:lifeskim |
| pubmed-article:17310371 | lifeskim:mentions | umls-concept:C0205217 | lld:lifeskim |
| pubmed-article:17310371 | lifeskim:mentions | umls-concept:C0596610 | lld:lifeskim |
| pubmed-article:17310371 | lifeskim:mentions | umls-concept:C0743925 | lld:lifeskim |
| pubmed-article:17310371 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:17310371 | pubmed:dateCreated | 2007-3-5 | lld:pubmed |
| pubmed-article:17310371 | pubmed:abstractText | Intrauterine growth in non-diabetic pregnancies is reported to be influenced by type 1 diabetes susceptibility genes. In particular, the high-risk HLA DR4_DQB1*0302 haplotype is associated with increased birthweight. The aim of this study was to determine whether HLA DR4 was associated with increased birthweight in a maternal diabetes environment and whether effects persisted during early childhood. | lld:pubmed |
| pubmed-article:17310371 | pubmed:language | eng | lld:pubmed |
| pubmed-article:17310371 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17310371 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:17310371 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17310371 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17310371 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:17310371 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:17310371 | pubmed:issn | 0012-186X | lld:pubmed |
| pubmed-article:17310371 | pubmed:author | pubmed-author:HummelMM | lld:pubmed |
| pubmed-article:17310371 | pubmed:author | pubmed-author:VoigtMM | lld:pubmed |
| pubmed-article:17310371 | pubmed:author | pubmed-author:BonifacioEE | lld:pubmed |
| pubmed-article:17310371 | pubmed:author | pubmed-author:HuppmannMM | lld:pubmed |
| pubmed-article:17310371 | pubmed:author | pubmed-author:MarienfeldSS | lld:pubmed |
| pubmed-article:17310371 | pubmed:author | pubmed-author:ZieglerA-GAG | lld:pubmed |
| pubmed-article:17310371 | pubmed:author | pubmed-author:KnopffAA | lld:pubmed |
| pubmed-article:17310371 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:17310371 | pubmed:volume | 50 | lld:pubmed |
| pubmed-article:17310371 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:17310371 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:17310371 | pubmed:pagination | 850-8 | lld:pubmed |
| pubmed-article:17310371 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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| pubmed-article:17310371 | pubmed:meshHeading | pubmed-meshheading:17310371... | lld:pubmed |
| pubmed-article:17310371 | pubmed:meshHeading | pubmed-meshheading:17310371... | lld:pubmed |
| pubmed-article:17310371 | pubmed:meshHeading | pubmed-meshheading:17310371... | lld:pubmed |
| pubmed-article:17310371 | pubmed:meshHeading | pubmed-meshheading:17310371... | lld:pubmed |
| pubmed-article:17310371 | pubmed:year | 2007 | lld:pubmed |
| pubmed-article:17310371 | pubmed:articleTitle | Fetal growth is increased by maternal type 1 diabetes and HLA DR4-related gene interactions. | lld:pubmed |
| pubmed-article:17310371 | pubmed:affiliation | Institute of Diabetes Research and Academic Hospital Schwabing, Kölner Platz 1, 80804, München, Germany. | lld:pubmed |
| pubmed-article:17310371 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:17310371 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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