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pubmed-article:17254356pubmed:abstractTextWith current treatment strategies, nearly half of all medulloblastoma (MB) patients die from progressive tumors. Accordingly, the identification of novel therapeutic strategies remains a major goal. Deregulation of c-MYC is evident in numerous human cancers. In MB, over-expression of c-MYC has been shown to correlate with anaplasia and unfavorable prognosis. In neuroblastoma--an embryonal tumor with biological similarities to MB--the quassinoid NBT-272 has been demonstrated to inhibit cellular proliferation and to down-regulate c-MYC protein expression.lld:pubmed
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pubmed-article:17254356pubmed:articleTitleAnti-proliferative activity of the quassinoid NBT-272 in childhood medulloblastoma cells.lld:pubmed
pubmed-article:17254356pubmed:affiliationNeuro-Oncology Program, University Children's Hospital, Zurich, Switzerland. Andre.vonBueren@kispi.unizh.ch <Andre.vonBueren@kispi.unizh.ch>lld:pubmed
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